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ResultsThe bibliometric analysis of COVID-19 and SuddenCardiacDeath highlights key research trends from 2020 to 2024, revealing a rapid surge in scientific output during the pandemic. Literature retrieved using the above keywords and published were included with a time limit from 1st January 2020 to 8th Aug 2024.ResultsThe
Background High-intensity physical activity has traditionally been discouraged in patients with hypertrophic cardiomyopathy due to concerns about triggering suddencardiacdeath. However, current guidelines adopt a more liberal stance, and evidence on risk factors for exercise-related suddencardiacdeath remains limited.
A new computational method developed by physicists can be used to estimate the risk of suddencardiacdeath from a one-minute heart rate measurement at rest. The study was carried out in interdisciplinary collaboration between cardiology and computational physics.
ABSTRACT Suddencardiacdeath (SCD) is a major public health challenge, resulting in significant loss of life each year. Effective prevention relies on identifying individuals at elevated risk; however, accurate prediction of SCD has proven difficult.
A study found people with schizophrenia have a 4-fold higher risk of suddencardiacdeath compared to the general population, underscoring the need for cardiometabolic care.
This cohort study evaluates the association of late gadolinium enhancement with suddencardiacdeath or relevant associated events among patients younger than 21 years with hypertrophic cardiomyopathy.
A recent major review of data published by The Lancet and led by Emory sports cardiologist Jonathan Kim, MD, shows that Black athletes are approximately five times more likely to experience suddencardiac arrest (SCA) and suddencardiacdeath (SCD) compared to white athletes, despite some evidence of a decline in rates of SCD overall.
Image courtesy of Yoshihiro Sobue from Fujita Health University mtaschetta-millane Tue, 07/30/2024 - 09:18 July 30, 2024 — Patients with congestive heart failure (CHF) having a compromised blood supply, are at greater risk of suddencardiacdeath (SCD).
Image courtesy of UCL Institute of Cardiovascular Science / James Tye milla1cf Tue, 12/19/2023 - 18:19 December 19, 2023 — A vest that can map the electrical activity of the heart in fine detail could potentially be used to better identify people at high risk of suddencardiacdeath , suggests a new study led by UCL researchers.
A reusable vest that can map the electrical activity of the heart in fine detail could potentially be used to better identify people at high risk of suddencardiacdeath, suggests a new study led by UCL researchers.
Middle-aged adults with long-term fluctuations in blood pressure may face a higher risk for suddencardiacdeath than their peers with more consistent blood pressure readings, a large new study suggests.
Suddencardiacdeath (SCD) remains a pressing health issue, affecting hundreds of thousands each year globally. The heterogeneity among SCD victims, ranging from individuals with severe heart failure to seemingly healthy individuals, poses a significant challenge for effective risk assessment.
(MedPage Today) -- The hunt for a signal of excess suddencardiacdeaths among young people after COVID-19 vaccination left Oregon health officials empty-handed, they reported. Investigators searched death certificates for Oregon residents 16.
Suddencardiacdeath is an important cause of mortality in patients with kidney failure undergoing renal replacement therapy, either hemodialysis or peritoneal dialysis. The risk factors associated with suddencardiacdeath in these patients only partly overlap with those in the general population.
Research Highlights: While the overall incidence of suddencardiacdeath among National Collegiate Athletic Association athletes decreased over. This abstract is simultaneously published with a full manuscript in the AHA’s flagship journal Circulation.
Nature Reviews Cardiology, Published online: 23 January 2024; doi:10.1038/s41569-024-00989-0 A molecular autopsy is undertaken in cases of suddencardiacdeath with no definitive cause found after conventional autopsy, with the aim of identifying a pathological genetic variant that could account for the death.
Approximately half the OCM cases were diagnosed with suddencardiacdeath (SCD), with significant differences. Macroscopically, OCM hearts had a “globoid” appearance with a thickened right ventricular outflow tract. Some OCM cases showed focal interstitial fibrosis in the left ventricle.
1-2 We aimed to determine the prevalence of intramyocardial adipose deposits in suddencardiacdeath (SCD) victims with CAD, and to analyze any associated characteristics.
Does use of left ventricular ejection fraction (LVEF) and/or other variables identify patients who benefit from defibrillator implantation to prevent suddendeath after acute myocardial infarction (MI)?
In this interview, Katherine Martinez, and Matthew Martinez MD, FACC, discuss From JACC: Return-to-Play for Elite Athletes With Genetic Heart Diseases Predisposing to SuddenCardiacDeath.
Persistent complex bereavement disorder occurs in 21% of first-degree relatives following suddencardiacdeath (SCD). The multifaceted needs of affected Chinese families are unknown.
Patients with congestive heart failure (CHF) having a compromised blood supply, are at greater risk of suddencardiacdeath (SCD). With an estimated incidence as high as 22% among these patients, current clinical guidelines recommend using implantable cardioverter defibrillators (ICDs) to help mitigate the risk of SCD.
The effects of SGLT2i on atrial fibrillation(AF), atrial flutter(AFL), composite AF/AFL, ventricular fibrillation(VF), ventricular tachycardia(VT), ventricular extrasystoles(VES), suddencardiacdeath(SCD) and composite VF/VT/SCD were evaluated. The mean age was 64.9 ± 9.4 ± 9.4 were male.
A new computational method developed by physicists at Tampere University can be used to estimate the risk of suddencardiacdeath from a one-minute heart rate measurement at rest. The study was carried out in interdisciplinary collaboration between cardiology and computational physics.
BACKGROUND:Declining cardiovascular mortality rates have been well-documented, yet temporal trends of suddencardiacdeath (SCD) in young individuals remain unclear. Circulation, Ahead of Print.
The use of the antipsychotic drugs quetiapine and haloperidol is associated with an increased risk of ventricular arrhythmias and suddencardiacdeath (SCD) caused by drug-induced QT prolongation, reports a new study.
Possible arrhythmic recurrences and the risk of suddencardiacdeath (SCD) in this setting are reasons for concern, and limited data have been published to guide clinical management of these patients. Background Ventricular arrhythmias (VAs) frequently occur in the acute phase of myocarditis.
Abstract The limited literature and increasing interest in studies on cardiac electrophysiology, explicitly focusing on cardiac ion channelopathies and suddencardiacdeath in diverse populations, has prompted a comprehensive examination of existing research.
Suddencardiacdeath (SCD) risk stratification is based on clinically recognized risk factors (RF), such as reduced left ventricular (LV) ejection fraction (EF), heart failure (HF), prior myocardial infarction (MI), and syncope. These RFs fail to capture the majority of SCDs.
Predicting suddencardiacdeath may be possible using artificial intelligence (AI) to analyze medical information in electronic health records, according to preliminary research.
Suddencardiacdeath represents a significant mortality factor in hypertrophic cardiomyopathy (HCM). Nevertheless, the validation of the existing risk prediction model in the pediatric population remains insufficiently explored.
Although varying definitions of ER, previous studies suggested that the prevalence varied between 2% to 31% in population-cohort studies and that ER patterns are associated with an increased risk of suddencardiacdeath (SCD). However, no consensus definition of ER existed until 2015.
It is the leading cause of suddencardiacdeath in young people and children, with an annual mortality rate of 1%. However, 10% to 20% of these patients have a significantly higher risk of suddencardiacdeath. Hypertrophic cardiomyopathy (HCM) is the most common genetic cardiovascular condition.
Suddencardiacdeath (SCD) has an estimated annual incidence of 1 per 1000, based on epidemiological studies from death certificates. However, up to 40% of suddendeaths have been determined to be caused by extracardiac causes in autopsy studies.1,2
Despite improved childhood survival in congenital heart disease (CHD) due to advances in management, late-onset suddencardiacdeath (SCD) from malignant ventricular arrhythmias remains a leading cause of mortality in adults with CHD (ACHD).
Diabetes mellitus (DM) confers an increased risk of suddencardiacdeath (SCD) independent of its associated cardiovascular comorbidities. Diabetes mellitus induces adverse structural, electrophysiological, and autonomic cardiac remodeling which can increase one’s risk of ventricular arrhythmias and SCD.
SCAD can cause acute coronary syndrome and myocardial infarction (MI), as well as suddencardiacdeath. Spontaneous coronary artery dissection (SCAD) is an uncommon condition which is increasingly recognized as a cause of significant morbidity.
A heart attack is a life-threatening condition, with patients remaining at risk of premature death long after the actual attack—50% to 60% of patients die subsequently as a result of suddencardiacdeath, triggered by cardiac arrhythmia.
ß-blockers and flecainide have been used for treatment of VT as were implantable cardioverter defibrillators (ICD) for prevention of suddencardiacdeath (SCD). Repolarization abnormalities similar to those observed in long QT syndrome (LQTS) are responsible for VT in ATS-1; therefore ATS-1 is sometimes referred to as LQTS 7.
Brugada syndrome (BrS) is associated with an increased risk of ventricular fibrillation (VF), leading to suddencardiacdeath. 1 However, variations in the efficacy of these drugs remain unclear, particularly regarding low-dose quinidine.
Patients with acute pulmonary embolism (PE) have a wide spectrum of clinical presentations, from incidental findings to suddencardiacdeath. Management and treatment recommendations in currently available guidelines vary according to patient risk and haemodynamic profile.
Suddencardiacdeath (SCD) as is defined as natural unexpected death in witnessed cases being <1 hour of symptoms and in unwitnessed cases as last seen alive <24hours (1). We have to be very precise in the definition of SCD as many suddendeaths may not be cardiac.
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