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Concerning EKG with a Non-obstructive angiogram. What happened?

Dr. Smith's ECG Blog

The commonest causes of MINOCA include: atherosclerotic causes such as plaque rupture or erosion with spontaneous thrombolysis, and non-atherosclerotic causes such as coronary vasospasm (sometimes called variant angina or Prinzmetal's angina), coronary embolism or thrombosis, possibly microvascular dysfunction.

Plaque 127
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Study Finds Olezarsen Cuts Triglyceride Levels by About Half

DAIC

As with elevated LDL-cholesterol, high levels of triglycerides and the lipid particles on which they are carried in the blood can contribute to the formation of “plaques” in the arteries that impede blood flow and can lead to heart attacks and strokes. Triglycerides store unused calories and provide energy to the body.

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Chest pain, resolved. Does it need emergent cath lab activation (some controversy here)? And much much more.

Dr. Smith's ECG Blog

A comparison of electrocardiographic changes during reperfusion of acute myocardial infarction by thrombolysis or percutaneous transluminal coronary angioplasty. Angiography : --Culprit for the patient's unstable angina/Wellen syndrome is a ruptured plaque in the mid LAD. --As Am Heart J. 2000;139:430–436.

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Abstract 4119169: Navigating Complexity: The Carlino Technique in Multivessel Percutaneous Coronary Intervention for Chronic Total Occlusion

Circulation

She was diagnosed with NSTEMI with a thrombolysis in myocardial infarction (TIMI) score of 5. Her EKG showed marked left-axis deviation, ST depressions in V2-V4, and RBBB. PCI, particularly with the Carlino technique, offers a reliable approach.

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An unusual query in Wellen’s syndrome ?

Dr. S. Venkatesan MD

It is generally believed it is more of a mechanical plaque lesion. RCA and LCX Wellens do occur, making this entity’s perceived unique importance less certain 3. How common is thrombosis in the culprit artery of Wellen syndrome ? However by no means, we can say thrombosis do not occur. Is there a benign face of Wellen syndrome ?

Anatomy 52
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A man in his 50s with acute chest pain and LVH

Dr. Smith's ECG Blog

Reperfusion of OMI indicates at least partial thrombolysis of occluding thrombus, but still unstable plaque rupture, which can reocclude at any moment. This case's first EMS ECG shows a rare example of LVH with active anterior and high lateral OMI. Comparison to prior ECG can be very helpful.

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90 year old with acute chest and epigastric pain, and diffuse ST depression with reciprocal STE in aVR: activate the cath lab?

Dr. Smith's ECG Blog

This has been termed a “STEMI equivalent” and included in STEMI guidelines, suggesting this patient should receive dual anti-platelets, heparin and immediate cath lab activation–or thrombolysis in centres where cath lab is not available.