MIBHS

MARCH 31, 2025

This blockage is often caused by a blood clot or the buildup of plaque in the coronary arteries, which supply the heart with oxygen-rich blood. Unlike a heart attack, cardiac arrest is an electrical malfunction that causes the heart to stop beating suddenly. Without immediate intervention, sudden cardiac death can occur within minutes.

Cardiac Arrest 52

Cardiac Arrest Heart Attack Heart attacks Chest Pain 52

Dr. Smith's ECG Blog

JUNE 23, 2024

His ECG at the accepting facility is shown below: Accepting facility ECG The team reviewed his angiography films with an interventionalist and thought they were suspicious for plaque rupture in LAD, but they were not confident.

Chest Pain 117

Chest Pain Cardiac Arrest Sudden Cardiac Death Blog 117

DAIC

OCTOBER 30, 2024

By adding AutoChamber AI to CAC scans and analyzing both coronary artery plaque calcification and cardiac chambers volume, HeartLung plans to introduce AI-CAC and AI-CVD which have the potential to advance preventive cardiology. There is a long list of diseases that can cause enlarged heart or enlarged cardiac chambers.

Cancer 45

Cancer Atrial Fibrillation Sudden Cardiac Death Heart Failure 45

Physiologically Speaking

FEBRUARY 28, 2024

A large amount of endurance-exercise-related deaths occurred due to a cardiac incident during or following activity. Vigorous exercise is known to increase the risk of sudden cardiac death. However, this increased risk manifests mostly among individuals with an underlying cardiac disease.

Exercise 98

Exercise Sudden Cardiac Death Physiology Congenital 98

DAIC

JANUARY 23, 2024

Atherosclerotic cardiovascular disease (ASCVD), caused by plaque buildup in arterial walls, is one of the leading causes of disability and death worldwide.1,2 alone, more than 800,000 of these people are at risk of MI and for approximately 200,000 of them, this may well be their second life-threatening cardiac event.

Cardiovascular Disease 111

Cardiovascular Disease Heart Attack Heart attacks Coronary Artery Disease 111

Dr. Smith's ECG Blog

OCTOBER 7, 2019

He was taken to the cath lab and underwent emergent intervention: Thrombotic stenosis of the proximal RCA (95% with evidence of plaque rupture) is the culprit for the patient's inferoposterior STEMI. He has a history of sudden cardiac death in his family. He was successfully stented. His troponin I peaked at 6.107 ng/mL.

Bradycardia 40

Bradycardia STEMI Cardiogenic Shock Ischemia 40

Dr. Smith's ECG Blog

JANUARY 9, 2023

No family history of sudden cardiac death, cardiomyopathy, premature CAD, or other cardiac issues. Only after her troponin peaked at 500,000 ng/L did she get her angiogram, which showed a 100% left main occlusion due to ruptured plaque. No similar symptoms in the past. RBBB, LAFB, and STE in I, aVL, V2 and V3.

Chest Pain 52

Chest Pain Aneurysm STEMI Thrombosis 52