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Background To investigate the correlation between lg (circSCMH1/miR-874) and acute coronary syndrome (ACS), acute myocardial infarction (AMI), and carotid plaque stability. Methods 701 patients were divided into stable coronary artery disease (SCAD), ACS, and control groups.
Here’s the angiogram of the RCA : No thrombus or plaque rupture in the RCA (or any coronary artery) was found. This MI wasn’t caused by a ruptured plaque of CAD - it was a coronary artery dissection of the RCA. In the absence of these factors it is termed spontaneous coronary artery dissection ( SCAD ). A study by Hassan et al.
If the arrest was caused by acute MI due to plaque rupture, then the diagnosis is MINOCA. Here is my comment on MINOCA: "Non-obstructive coronary disease" does not necessarily imply "no plaque rupture with thrombus." They often cannot even be recognized as culprits, as fissured or ulcerated plaque. What is Type 2 MI?
This was ruptured plaque with thrombus. It was not SCAD (coronary dissection) Highest troponin I was 37,000 ng/L, but it was not measured to peak. And almost all of them could be detected by bedside ultrasound. It is not a waste of time to use bedside ultrasound to look for dissection 3. Ultrasound Med.
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