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Factors associated with high-intensity physical activity and sudden cardiac death in hypertrophic cardiomyopathy

Heart BMJ

Background High-intensity physical activity has traditionally been discouraged in patients with hypertrophic cardiomyopathy due to concerns about triggering sudden cardiac death. However, current guidelines adopt a more liberal stance, and evidence on risk factors for exercise-related sudden cardiac death remains limited.

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The Risks and Benefits of 'Too Much' Exercise

Physiologically Speaking

” While an awe-inspiring anecdote, our knowledge of human limits, physiology, and the sheer number of participants in endurance sports is enough evidence to conclude that running a marathon won’t kill you. Physiologically Speaking is a reader-supported publication. Pheidippides’ story is provocative.

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The autonomic nervous system and the origins of neurocardiology

Journal of Cardiovascular Electrophysiology

Neurocardiology, perhaps initially more directed towards our understanding of sudden cardiac death, ultimately embraced an even significantly more complex scheme of local circuit neurons and near-endless loops of interconnecting neurons in the heart.

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Development of the NOCSAE Standard to Reduce the Risk of Commotio Cordis

Circulation: Arrhythmia and Electrophysiology

BACKGROUND:Commotio cordis, sudden cardiac death (SCD) caused by relatively innocent impact to the chest, is one of the leading causes of SCD in sports. A physiological commotio cordis model was utilized to assess variables that predicted for SCD. In the third phase, this model was calibrated and validated.

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What kind of AV block is this? And why does she develop Ventricular Tachycardia?

Dr. Smith's ECG Blog

The granulomatous inflammation affects the heart, causing an infiltrative cardiomyopathy The most common manifestations of cardiac sarcoidosis are atrioventricular (AV) block and ventricular tachyarrhythmias (VT). SCD (sudden cardiac death) from sarcoid heart disease is thought to be caused by either from high-grade AV block or VT.

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Why ICDs are less effective in Non Ischemic DCM ?

Dr. S. Venkatesan MD

We wish, our understanding about cardiac contractile physiology is deep and nearly complete. Bardy GH, Lee KL, Mark DB, Poole JE, Sudden Cardiac Death in Heart Failure Trial (SCD-HeFT) Investigators. Heart is an irreversibly coupled electro-mechanical organ , right from the fetal days until the final heart beat.

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A 40-Something male with a "Seizure," Hypotension, and Bradycardia

Dr. Smith's ECG Blog

He has a history of sudden cardiac death in his family. This also confirms right ventricular infarction (RV MI) A follow up TTE demonstrated a normal LVEF with a “regional wall motion abnormality-posterolateral hypokinetic mild, probable.” His troponin I peaked at 6.107 ng/mL. Learning Points : 1.