Physiologically Speaking

JANUARY 5, 2024

Welcome to the Physiology Friday newsletter. Physiologically Speaking is a reader-supported publication. One of the biggest risk factors for CVD development is the buildup of plaque in the coronary arteries (the arteries surrounding the heart that provide it with its own blood supply).

Plaque 119

Plaque Physiology Exercise Cholesterol 119

Dr. Smith's ECG Blog

JANUARY 5, 2024

Although it is statistically unlikely, multiple plaque ruptures are possible. On intravascular ultrasound (IVUS), the mid RCA plaque was described as "cratered, inflamed, and bulky," and the OM plaque was described as "bulky with evidence of inflammation and probably ulceration." Additional findings: No ST elevation."

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Angina Plaque Document Stent 121

Stroke: Vascular and Interventional Neurology

NOVEMBER 9, 2023

The deformable gel constituted an 8mm long replaceable stenotic segment at the level of M1‐MCA simulating an atherosclerotic plaque with a 0.5mm internal diameter. mL/min.ConclusionThe developed ICAD model is anatomically accurate and offers realistic physiological and procedural features.

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Stenosis Angioplasty Anatomy Plaque 40

Dr. Sanjay Gupta

MARCH 6, 2022

It is also unique because it works using the Doppler effect, you can get not only an anatomical evaluation of the heart but also physiological assessment. An echo is easy to do, risk free and easily accessible. It is operator dependant and requires specialised machinery The images you get may vary from patient to patient.

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Heart attacks Heart Attack Heart Rhythm Exercise 52

Dr. Smith's ECG Blog

NOVEMBER 26, 2018

The axiom of "type 1 (ACS, plaque rupture) STEMIs are not tachycardic unless they are in cardiogenic shock" is not applicable outside of sinus rhythm. This case represents the same physiologic event as OMI in terms of the result on the myocardium, therefore with identical ECG features, however there may not be ACS!

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Ischemia Atrial Flutter Arrhythmia Tachycardia 52

Dr. Smith's ECG Blog

OCTOBER 7, 2019

He was taken to the cath lab and underwent emergent intervention: Thrombotic stenosis of the proximal RCA (95% with evidence of plaque rupture) is the culprit for the patient's inferoposterior STEMI. He was successfully stented.

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Bradycardia STEMI Cardiogenic Shock Ischemia 40

Dr. S. Venkatesan MD

DECEMBER 8, 2024

The reason being, there is a huge healthy population ( with zero risk factor) , but showing insignificant or minimal coronary plaques. In adults, some of these streaks become prominent locally and turn out to be plaque. The argument for intensive statin therapy is to stabilize these plaques. Reference 1. J Am Coll Cardiol.

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Plaque Risk Factors Document Physiology 52