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Introduction Spontaneous coronary artery dissection (SCAD) is a non-traumatic and non-iatrogenic separation of the coronary arterial wall. Results 14 studies with 2,145 females in the generative period with ACS caused by SCAD were analyzed. The most common riskfactor was previous smoking history in 24.9% of patients.
The patient was previously healthy, with no atherosclerotic riskfactors, and developed chest pain after an episode of stress. Here is the post shock ECG: Cardiology was called stat for ischemic VT, query SCAD vs thrombotic occlusion vs coronary vasospasm. Echo showed EF of 50% with akinetic apex.
She had zero CAD riskfactors. What is Spontaneous Coronary Artery Dissection (SCAD)? I asked Angie Lobo ( [link] ), a third year intermal medicine resident at Abbott Northwestern Hospital (and Minneapolis Heart Institute) and an aspiring cardiologist, to write a couple paragraphs on SCAD.
Background Spontaneous coronary artery dissection (SCAD) and Takotsubo syndrome (TTS) constitute two common causes of nonatherosclerotic acute cardiac syndrome particularly frequent in women. Currently, there is no information comparing long-term clinical outcomes in unselected patients with these conditions. In-hospital events (43.3%
Soviet biologist Trofim Lysenko famously rejected the objective reality of Mendelian genetics because it clashed with the Marxist philosophy that the environment, not genetics, was the primary determinant of outcomes. Women also had more cardiovascular riskfactors, including hypertension (66.6% years of age versus 59.0±8.4
Sex differences exist in the prevalence and effect of cardiovascular riskfactors. For example, women with history of traditional cardiovascular riskfactors including hypertension, tobacco use, and diabetes carry a higher risk of major cardiovascular events and mortality when compared to men.
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