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One of my most talented readers is a health care assistant (a nursing assistant) who has taken a keen interest in ECGs. Was there pulmonary edema? Trop T now very high, well into the range one sees with a STEMI; very unusual in type II MI. And they teach me a lot. He can beat nearly anyone. You don't have to be a genius.
Without seeing the patient, my interpretation of the first ECG was: likely normal variant ST-elevation (early repolarization), with a small possibility of pericarditis, and almost no possibility of acute coronary occlusion (STEMI). and therefore highly unlikely to be STEMI. The ST/T ratio in V6, however, is slightly greater.
Bedside ultrasound showed no effusion and moderately decreased LV function, with B-lines of pulmonary edema. Here is his ED ECG: There is obvious infero-posterior STEMI. What are you worried about in addition to his STEMI? to greatly decrease risk (although in STEMI, the optimal level is about 4.0-4.5 Is 40 mEq too much?
50% of LAD STEMIs do not have reciprocal findings in inferior leads, and many LAD OMIs instead have STE and/or HATWs in inferior leads instead. The ECG easily meets STEMI criteria in all leads V2-V6, as well. CT angiogram chest: no aortic dissection or pulmonary embolism. 24 yo woman with chest pain: Is this STEMI?
The paramedics diagnosis was "Possible Anterolateral STEMI." Chest X-ray also showed pulmonary edema. More proof that a huge STEMI may have normal or near normal initial troponin. Primary VF in this study refers to fibrillation occurring in the absence of shock or pulmonary edema. She was given 2 mg Magnesium.
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