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PAD is a serious, progressive cardiovascular disease primarily caused by a buildup of fatty plaque in the blood vessels, or atherosclerosis. This plaque narrows the blood vessels and reduces blood flow to the legs and feet, which may significantly impair physical function, walking performance and quality of life.
It’s a multidisciplinary approach involving nurses, doctors, physiotherapists, dieticians, psychologists and many others. Just because you have heart disease or have had a heart attack does not mean there is a lot that can be done. Cardiac Rehabilitation. This is a program that starts the day of your heart attack.
A 69‐year‐old woman with a history of lung cancer, hypertension, chronic tobacco use, atherosclerosis, and known calcified plaque at the left carotid bifurcation on dual antiplatelet therapy presented with acute onset of expressive aphasia and right hemiparesis due to acute left CCAO. Post‐operatively, he had evacuation of neck hematoma.
David Didlake Acute Care Nurse Practitioner Firefighter / Paramedic (ret) @DidlakeDW Expert commentary and peer review by Dr. Steve Smith [link] @smithECGBlog A 57 y/o Female with PMHx HTN, HLD, DM, and current use of tobacco products, presented to the ED with chest discomfort. This results in Type I MI. Severe Hypoxia b.
Angiogram: Severe coronary artery calcification Moderate to severe distal small vessel disease mainly seen in RPL1, 2 Otherwise, Mild plaque, no angiographically significant obstructive coronary artery disease. This would be the likely source of the VT. PEARL #1 — Remember statistical likelihoods.
It had started just after nursing her newborn, about an hour prior, and she described it as a severe non-pleuritic “pressure” radiating to the back. Here’s the angiogram of the RCA : No thrombus or plaque rupture in the RCA (or any coronary artery) was found. This is written by Brooks Walsh.
David Didlake Firefighter / Paramedic Acute Care Nurse Practitioner @DidlakeDW Peer review provided by Dr. Steve Smith [link] @SmithECGblog A 72 y/o Male experiences a syncopal episode while seated. 2] Although the clinical context in today’s case does not fit these descriptors for Type I OMI (e.g.
The coronary angiogram revealed no critical stenosis, or acute plaque ulceration. Surawicz and Knilans report that intense catecholamine surge, or severe maladjustment of the autonomic nervous system, can manifest “cerebral T waves” in the absence of an acute intracranial process. Furthermore, pertinent electrolyte values (e.g.
This case reminds me of this 27 year old totally healthy nurse who was previously healthy, presented with acute pulmonary edema and the below ECG that is diagnostic of proximal LAD occlusion, and was dismissed because of her age. She died before she could get a heart transplant.
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