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Long-term transcranial ultrasound stimulation regulates neuroinflammation to ameliorate post-myocardial infarction cardiac arrhythmia and remodeling

HeartRhythm

Sympathetic overactivation and neuroinflammation in the paraventricular nucleus (PVN) are crucial factors in post-myocardial infarction (MI) cardiac remodeling and ventricular arrhythmias (VAs). Meanwhile, the cGAS-STING pathway has shown potential to ameliorate neuroinflammatory response.

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Advanced ultrasound cannot discern who would benefit from beta-blocker treatment after myocardial infarction

Medical Xpress - Cardiology

A new study by researchers at Karolinska Institutet and Sdersjukhuset shows that advanced ultrasound imaging analyses cannot identify patients who would benefit from beta-blocker treatment after a myocardial infarction.

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Low-intensity focused ultrasound modulation of the paraventricular nucleus to prevent myocardial infarction-induced ventricular arrhythmia

HeartRhythm

Our previous study showed that light-emitting diode (LED) modulation of hypothalamic paraventricular nucleus (PVN), which is the control center of the sympathetic nervous system, might attenuate neuroinflammation in PVN and prevent ventricular arrhythmias (VAs) after myocardial infarction (MI).

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Imaging patients with myocardial infarction with non-obstructive coronary arteries (MINOCA)

Heart BMJ

Myocardial infarction with non-obstructive coronary arteries (MINOCA) defines a heterogeneous group of atherosclerotic and non-atherosclerotic conditions, causing myocardial injury in the absence of obstructive coronary artery disease.

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Missed myocardial infarction with subsequent cardiac arrest

Dr. Smith's ECG Blog

An emergency cardiac ultrasound could be very useful. The upright portion of the T-wave in aVF is very large compared to the QRS size. These findings mandate that the patient at least get serial ECGs. If these remain unchanged, then serial troponins.

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Case Report: Extended cardiopulmonary resuscitation in sudden cardiac arrest after acute myocardial infarction

Frontiers in Cardiovascular Medicine

However, the patient's cardiac Doppler ultrasound indicated poor cardiac contractions, and extracorporeal membrane oxygenation (ECMO) was started immediately. We administered adrenaline for cardiac excitation, dopamine for maintained blood pressure, sodium bicarbonate to correct the acidosis, and multiple electric defibrillations.

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Dynamic OMI ECG. Negative trops and negative angiogram does not rule out coronary ischemia or ACS.

Dr. Smith's ECG Blog

Bedside cardiac ultrasound with no obvious wall motion abnormalities. Thus, it has recently become generally accepted that most plaque ruptures resulting in myocardial infarction occur in plaques that narrow the lumen diameter by 40% of the arterial cross section may be involved by plaque. He was started on nitro gtt.

Ischemia 121