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ECG Guru
JULY 26, 2023
76-year-old man, with a history of inferior wall myocardial infarction. In this ECG, at the beginning, the last part of a wide-complex tachycardia is visible. After 2 sinus beats, another wide-complex tachycardia begins (with the same QRS configuration).
Medical Xpress - Cardiology
OCTOBER 8, 2024
A multicenter study has described and validated a new strategy for guiding ablation procedures in patients with complex tachycardias. This type of procedure is frequently used to treat ventricular tachycardias originating in areas affected by scarring after a myocardial infarction.
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ECG Guru
DECEMBER 18, 2023
If a wide complex tachycardia occurs, the probability is very high that it is a ventricular tachycardia (approx. 80%, in patients with a previous myocardial infarction (.) Here we see a broad complex tachycardia that looks like an RBBB + LAFB, which is regular. In this constellation, 3 causes must be considered: 1.
The New England Journal of Medicine
OCTOBER 30, 2024
Regular tachycardia (124 beats per min), diaphoresis, and rales were present. A 69-year-old man with a history of MI and cardiomyopathy presented with 2 days of dyspnea. A diagnosis was made.
HeartRhythm
APRIL 30, 2024
Wide complex tachycardias (WCT) in patients after myocardial infarction (MI) are often VT. A correct diagnosis is vital.
ECG Guru
JULY 31, 2023
Broad complex tachycardia in a 78-year-old patient with coronary heart disease (CHD) and an old inferior myocardial infarction. Why is this a ventricular tachycardia (VT) and not a supraventricular tachycardia (SVT) with aberrant conduction? What other clues are there?
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All About Cardiovascular System and Disorders
APRIL 17, 2024
Ventricular tachycardia is a potentially life threatening cardiac arrhythmia. On the ECG, ventricular tachycardia can be defined as three or more ventricular ectopic beats occurring in a sequence at a rate more than 100 per minute. Another rare form of ventricular tachycardia is bidirectional ventricular tachycardia.
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ECG Guru
DECEMBER 18, 2023
If a wide complex tachycardia occurs, the probability is very high that it is a ventricular tachycardia (approx. 80%, in patients with a previous myocardial infarction (.) Here we see a broad complex tachycardia that looks like an RBBB + LAFB, which is regular. In this constellation, 3 causes must be considered: 1.
HeartRhythm
JANUARY 16, 2025
A 64-year-old male with recurrent ventricular tachycardia (VT) was referred for repeat ablation. His medical history included prior myocardial infarction and cardiac magnetic resonance imaging demonstrated basal inferior and anteroseptal subendocardial scar with overall preserved left ventricular function.
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Frontiers in Cardiovascular Medicine
MARCH 3, 2024
Background Early ventricular tachycardia/fibrillation (VT/VF) in patients with ST-elevation myocardial infarction (STEMI) has higher morbidity and mortality. This study examines gender-differentiated risk factors and underlying mechanisms for early onset VT/VF in STEMI.
Dr. Smith's ECG Blog
SEPTEMBER 5, 2024
This is a value typical for a large subacute MI, n ormal value 48 hours after myocardial infarction is associated with Post-Infarction Regional Pericarditis ( PIRP ). Sinus tachycardia has many potential causes. This is especially true for the elderly patient with sinus tachycardia. Hammill SC. Edwards WD.
Frontiers in Cardiovascular Medicine
APRIL 8, 2025
The principal clinical manifestation of thrombophilia is venous thromboembolism, which is also markedly linked to arterial thrombosis, including myocardial infarction. This report presents the case of a young man who suffered an acute myocardial infarction as a result of thrombophilia.
All About Cardiovascular System and Disorders
APRIL 23, 2024
Three or more ventricular beats in a row at a rate above 100 per minute is termed ventricular tachycardia. Ventricular tachycardia lasting more 30 seconds or requiring termination earlier due to hemodynamic compromise is called sustained ventricular tachycardia. Either case, the treatment is ablation of the right bundle.
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Dr. Smith's ECG Blog
FEBRUARY 8, 2015
They had already cardioverted at 120 J, then 200 J, which resulted in the following: Ventricular Tachycardia They then cardioverted at 200 J which r esulted in the same narrow complex rhythm shown above, at 185 beats per minute. This would treat both SVT or sinus tachycardia. I suggested esmolol if the heart rate did not improve.
52 HeartRhythm
APRIL 30, 2024
Mapping data in post-myocardial infarction (MI) patients describe ventricular tachycardia (VT) re-entry circuits of slow VT, that are typically related to transmural scars. Fast VTs with cycle lengths close to the refractory period may be related to functional re-entry in the scar border zone (BZ).
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Dr. Smith's ECG Blog
AUGUST 13, 2020
Alternatively , it is someone who has an old myocardial infarction and is now very sick with something else. The patient spontaneously converted back to sinus tachycardia. They agreed that the ECG findings were due only to old MI and tachycardia. Later, I obtained more clinical history. Looks like atrial fibrillation.
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Frontiers in Cardiovascular Medicine
JANUARY 25, 2024
Perioperative myocardial injury is a relatively common complication after noncardiac surgery associated with significant morbidity and mortality. It is frequently driven by physiologic factors such as hypotension, tachycardia, and anemia.
Journal of Cardiovascular Electrophysiology
DECEMBER 10, 2023
The activation signal, or signature, defined as the cumulative number of recording sites that have activated per millisecond, was utilized to segment each re-entrant ventricular tachycardia (VT) circuit into inner and outer circuit pathways, and as an estimate of best ablation lesion location to prevent VT.
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Dr. Smith's ECG Blog
MARCH 1, 2024
The tachycardia was gone by the time paramedics arrived. He was unconscious for 8 minutes and slowly awoke in the ambulance, complaining of nausea only. First responders found him to be very tachycardic , confused, perserverating and with no memory of the event. There was tongue biting. Lightheadedness continued. Interpretation.
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Dr. Smith's ECG Blog
NOVEMBER 22, 2023
This is ischemic ST depression, and could be due to increasing tachycardia, with a heart rate over 130, but that is unlikely given that the patient is now complaining of crushing chest pain and that there was tachycardia all along. There is widespread ST depression. Figure-1: Comparison of the first 2 ECGs in today's case.
119 
Frontiers in Cardiovascular Medicine
JULY 8, 2024
BackgroundHuman pluripotent stem cell-derived cardiomyocytes (hPSC-CMs) show tremendous promise for cardiac regeneration following myocardial infarction (MI), but their transplantation gives rise to transient ventricular tachycardia (VT) in large-animal MI models, representing a major hurdle to translation.
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Cardiomatics
APRIL 5, 2024
Sinus tachycardia – sinus rhythm above 100 bpm is a sinus tachycardia. Ventricular tachycardia – more than 7 consecutive complexes originating from ventricles at a rate of > 100 bpm. Supraventricular tachycardia – more than 7 consecutive complexes of supraventricular beats at a rate of > 100 bpm.
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HeartRhythm
OCTOBER 21, 2024
Different imaging modalities have been used to image and display scar tissue accelerating substrate identification and streamlining mapping and ablation of ventricular tachycardias (VTs). Cardiac magnetic resonance imaging (CMR) is referred as the gold standard for identifying scar tissue especially after myocardial infarction or injury1.
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All About Cardiovascular System and Disorders
NOVEMBER 16, 2023
In that study commonest ECG abnormalites were QTc prolongation followed by brady/tachycardia and then ST segment deviations [3]. ECG changes resembling ST elevation myocardial infarction has also been described after traumatic intracranial hemorrhage [4]. In a study of 204 subjects, 31% had troponin elevation. N Engl J Med.
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HeartRhythm
SEPTEMBER 10, 2023
The classical view that scarred, fibrotic myocardium in patients with myocardial infarction (MI) is the basis for ventricular tachycardia by producing a critical activation delay in surviving myocardial strands1 was recently challenged by Sung et al.2
Dr. Smith's ECG Blog
AUGUST 6, 2024
The ECG is diagnostic of occlusion myocardial infarction (OMI). I would have favored emergent coronary CT angiogram had I cared for this patient. == Learning points: Diagnosis of occlusion myocardial infarction (OMI) can be extremely difficult and takes time and effort to learn well.
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Dr. Smith's ECG Blog
JUNE 17, 2018
There is sinus tachycardia. Sinus tachycardia, which exaggerates ST segments and implies that there is another pathology. I have always said that tachycardia should argue against acute MI unless there is cardiogenic shock or 2 simultaneous pathologies. Here is that ECG: What do you think? See Table for diagnostic utility.
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Dr. Smith's ECG Blog
JANUARY 1, 2013
Notice there is tachycardia. I have warned in the past that one must think of other etiologies of ischemia when there is tachycardia. In this case, the patient had failed to take his atenolol in the AM and was having reflex tachycardia in addition to ACS. But we are now concerned with the precordial leads. BP was 160/100.
52 
Stroke Journal
JANUARY 30, 2025
Primary outcome was risk of ischemic stroke while secondary outcomes were all-cause mortality (ACM), heart failure, acute myocardial infarction (AMI), major adverse cardiovascular events (MACE), atrial fibrillation (AF), and ventricular tachycardia (VT).Result:After
40 
Dr. Smith's ECG Blog
JUNE 17, 2017
There is sinus tachycardia. Because of the high troponin, echocardiography was done and showed a wall motion abnormality in the anterior, anterolateral, and apical walls, consistent with LAD myocardial infarction. I saw this as I was reading a large a stack of ECGs: What do you think? The QTc is 462 ms. It was stented.
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EMS 12-Lead
MARCH 19, 2023
He denied any known medical history, specifically: coronary artery disease, hypertension, dyslipidemia, diabetes, heart failure, myocardial infarction, or any prior PCI/stent. Breath sounds were clear in all lung fields. No appreciable skin pallor. He reported to be a social drinker, but used tobacco products daily.
All About Cardiovascular System and Disorders
APRIL 13, 2024
ECG in a person with persistent anginal pain for the past several hours showing significant ST segment depression anterolateral leads along with sinus tachycardia. Value of the electrocardiogram in localizing the occlusion site in the left anterior descending coronary artery in acute anterior myocardial infarction.
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Dr. Smith's ECG Blog
NOVEMBER 4, 2022
New insights into the use of the 12-lead electrocardiogram for diagnosing acute myocardial infarction in the emergency department. Here is the abstract: Background Identification of ST elevation myocardial infarction (STEMI) is critical because early reperfusion can save myocardium and increase survival.
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Dr. Smith's ECG Blog
DECEMBER 14, 2016
Acute myocardial injury: Is it myocardial infarction, or perhaps myocarditis? The patient had no hypertension, no tachycardia, a normal hemoglobin, no drug use, no hypotension/shock, no murmur of aortic stenosis. If it is MI, is it type 1 or type 2? Is it STEMI or NonSTEMI? Is it acute persistent occlusion?
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Dr. Smith's ECG Blog
MAY 19, 2022
The important point for our purposes is that they do no represent myocardial infarction. T-wave inversions and dynamic ST elevation Tachycardia, hyperthyroid, and ST elevation. Dr. Smith note: I wouldn't necessarily consider this ENTIRELY "benign." Indeed, they have a higher incidence of structural problems. What is it?
52 
Frontiers in Cardiovascular Medicine
FEBRUARY 24, 2025
IntroductionAcute coronary syndrome refers to a group of diseases characterized by sudden, decreased blood supply to the heart muscle that results in cell death, also known as acute myocardial infarction. The majority of patients (67.9%) have been diagnosed with ST- Elevated Myocardial Infarction and were classified as Killip class I.
98 
Dr. Smith's ECG Blog
JULY 21, 2019
I do not think this is acute occlusion myocardial infarction (OMI). I learned more about the history: 30-something African American with 5-7days of sharp R-sided shoulder/scapula/chest discomfort, presented with sinus tachycardia. Get an emergent contrast echocardiogram. These are reasons why it does not look like OMI: 1.
52 
Dr. Smith's ECG Blog
MAY 10, 2023
ng/mL This single initial troponin at this level, in the context of chest pain, is high enough to be diagnostic of acute myocardial infarction. That said, against acute PE — is the inconstancy of this patient's symptoms — the lack of tachycardia — and the lack of any other ECG signs of acute RV strain.
Dr. Smith's ECG Blog
APRIL 17, 2023
In addition to sinus tachycardia, the only abnormalities listed by the computer were "low voltage, precordial leads" and "anteroseptal infarct, old.Q Next trop 85,528 ng/L (consistent with a massive myocardial infarction), none further measured. 4) There is well formed J-point notching. Case 2: What do you think?
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Dr. Smith's ECG Blog
MARCH 11, 2019
Cardiac Troponin Changes to Distinguish Type 1 and Type 2 Myocardial Infarction and 180-Day Mortality Risk. In such cases, it is common for tachycardia to exaggerate the ST Elevation And, in fact, there was no new acute MI at this visit - troponins did not rise again. Murakami M. Acute Cardiovascular Care 2014;3(4):317-325.
52 Dr. Smith's ECG Blog
NOVEMBER 19, 2016
myocardial infarction), arrhythmias, valvular pathology, shunts, or outflow obstructions. Hypotension may of course be a result of a brady- or tachydysrhythmia. 2) Hypoxia, including poisons of oxidative phosphorylation such as HS, CO, CN. Fundamentally, cardiogenic shock is an issue of decreased cardiac output.
40 
All About Cardiovascular System and Disorders
MARCH 17, 2024
Then, why is it mentioned that, implanting a defibrillator soon after an acute myocardial infarction, in those with left ventricular dysfunction and prone for ventricular arrhythmias and sudden cardiac death, is not useful? Then, why is it mentioned that, an implantable defibrillator, is not useful, soon after myocardial infarction?
HeartRhythm
JANUARY 26, 2024
Sympathetic nervous system hyperactivity plays a major role in the pathogenesis of ventricular arrhythmias following myocardial infarction (MI).1,2 1,2 The stellate ganglia are an important nexus point for sympathetic innervation to the heart.3
80 
Circulation
NOVEMBER 11, 2024
Background:Ischemia-related ventricular arrhythmia is the leading cause of death in patients after acute myocardial infarction (AMI). To test the ventricular arrhythmogenesis post-AMI, we measured ventricular tachycardia/fibrillation (VT/VF) from 24-hour telemetry ECG recording in conscious rats.
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