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PO-02-101 SILENT MYOCARDIAL INFARCTION AND DILATED CARDIOMYOPATHY AMONG COUNTYWIDE SUDDEN CARDIAC DEATHS

HeartRhythm

Sudden cardiac death (SCD) risk stratification is based on clinically recognized risk factors (RF), such as reduced left ventricular (LV) ejection fraction (EF), heart failure (HF), prior myocardial infarction (MI), and syncope. These RFs fail to capture the majority of SCDs.

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Spontaneous Coronary Artery Dissection: A Focus on Post-Dissection Care for the Vascular Medicine Clinician

Frontiers in Cardiovascular Medicine

SCAD can cause acute coronary syndrome and myocardial infarction (MI), as well as sudden cardiac death. Spontaneous coronary artery dissection (SCAD) is an uncommon condition which is increasingly recognized as a cause of significant morbidity.

SCAD 137
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Predicting Sudden Cardiac Death After Acute MI

American College of Cardiology

Does use of left ventricular ejection fraction (LVEF) and/or other variables identify patients who benefit from defibrillator implantation to prevent sudden death after acute myocardial infarction (MI)?

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Intramyocardial fibrofatty deposits in ischemic sudden cardiac death

HeartRhythm

Recent studies suggest that intramyocardial adipose tissue facilitates ventricular tachycardia (VT) in subjects with coronary artery disease (CAD) and prior myocardial infarction (MI) scar.1-2

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Predictive Effect of Atypical Right Bundle‐Branch Block on In‐Hospital Sudden Cardiac Death and Cardiac Rupture and Long‐Term Prognosis in Patients With Acute Myocardial Infarction Who Underwent Percutaneous Coronary Intervention

Journal of the American Heart Association

ConclusionsARBBB is a predictor of inhospital SCD, CR, and 2year major adverse cardiovascular and cerebrovascular events in patients with firstepisode acute myocardial infarction undergoing percutaneous coronary intervention with a drugeluting stent.

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Why is an Implantable Defibrillator NOT Useful Soon After Myocardial Infarction?

All About Cardiovascular System and Disorders

Then, why is it mentioned that, implanting a defibrillator soon after an acute myocardial infarction, in those with left ventricular dysfunction and prone for ventricular arrhythmias and sudden cardiac death, is not useful? Mostly for protection against sudden cardiac death. That is one reason.

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The Irate Stellate Ganglion: IL-6 in Neuroinflammation-induced Ventricular Arrhythmias

HeartRhythm

Sympathetic nervous system hyperactivity plays a major role in the pathogenesis of ventricular arrhythmias following myocardial infarction (MI).1,2 1,2 The stellate ganglia are an important nexus point for sympathetic innervation to the heart.3