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IntroductionCardiovascular diseases include myocardialinfarction, a high mortality disease. Myocardialinfarction patients are becoming younger, typically defined as patients under 45 years of age. We searched the WoSCC between 1980 and 2023 for research papers and reviews on myocardialinfarction in youth.
The development of coronary care units is often credited for a dramatic decline in mortality rates after acute myocardialinfarction throughout the 1960s. As the underlying patient population became progressively sicker, changes in organizational structure, staffing, care delivery, and training paradigms lagged.
However, studies on diagnostic markers related to cellular senescence in acute myocardialinfarction (AMI) have not been reported. IntroductionCellular senescence can cause heart failure.
Background Early prediction of heart failure (HF) after acute myocardialinfarction (AMI) is essential for personalized treatment. Individual risk assessment was performed using SHAP plots and waterfall plot analysis.ConclusionsOur research demonstrates the potential of ML methods in the early prediction of HF risk in AMI patients.
ObjectiveThis study aimed to evaluate the correlation between aerobic exercise intensity based on the 6min walk test (6MWT) and the anaerobic threshold (AT)-based equivalent in patients with acute myocardialinfarction (AMI).
Yet, the impact of GLP-1RA treatment before ST-segment elevation myocardialinfarction (STEMI) on. Glucagon-like peptide-1 receptor agonist (GLP-1RA) treatment reduces cardiovascular events in type 2 diabetes.
However, whether immediate blood glucose and FIB levels affect coronary blood flow during primary percutaneous coronary intervention (PCI) remains unclear.ObjectiveTo explore the correlation between admission blood glucose (ABG), fibrinogen (FIB) and slow blood flow during primary PCI for acute ST segment elevation myocardialinfarction (STEMI).MethodsA
Two researchers independently screened, extracted data, and assessed quality using the Newcastle-Ottawa Scale (NOS) and Revman5.3. Meta-analysis with Stata 16.0 calculated the combined Odd ratio (OR) for WHR and MI risk.
ST-elevation myocardialinfarction (STEMI) is a critical cardiovascular emergency characterized by acute coronary artery occlusion and subsequent myocardial injury. Finally, the clinical evidence is critically assessed, existing controversies are analyzed, and directions for future research are proposed.
Stress hyperglycemia, which is associated with poor prognosis in patients with acute myocardialinfarction (AMI), can be determined using the stress hyperglycemia ratio (SHR). Impaired left ventricular functio.
The aim of this study was to investigate the difference between perceived and calculated diabetes risks among post-myocardialinfarction (AMI) patients using the Finnish Diabetes Risk Score (FINDRISC).
Cardio-cerebral infarction (CCI) is a rare clinical syndrome characterized by the simultaneous or sequential occurrence of acute myocardialinfarction (AMI) and acute ischemic stroke (AIS).
It is well-known that systemic inflammation plays a crucial role in the pathogenesis and prognosis of acute myocardialinfarction (AMI). The systemic immune-inflammation index (SII, platelet × neutrophil/lymph.
The impact of sodiumglucose cotransporter-2 (SGLT2) inhibitors on mortality following myocardialinfarction (MI) remains uncertain. Additionally, the role of type 2 diabetes mellitus (T2DM) and heart failure.
(MedPage Today) -- For the first time, intravascular imaging-guided coronary stenting was associated with better survival and reduced myocardialinfarction (MI) in a meta-analysis powered to detect these benefits against angiography, researchers.
The objective was to assess the clinical efficacy of long non-coding RNA (lncRNA) alpha-2-macroglobulin-antisense 1 (A2M-AS1) in acute myocardialinfarction (AMI).
The paucity of data necessitates further research to elucidate the pathophysiological connection between depression and cardiac diseases in the presence of N-terminal pro-brain natriuretic peptide (NT-proBNP). IntroductionDepression is a significant comorbidity linked to poor subjective and objective health outcomes in cardiac patients.
Diabetes mellitus (DM) and coronary microvascular dysfunction (CMD) increase the risk of adverse cardiac events in patients with non-ST-segment elevation myocardialinfarction (NSTEMI). This study aimed to eva.
When the heart is injured, as in a myocardialinfarction, the damaged heart muscle cannot regenerate—instead, scar tissue forms. Cardiomyocytes, the heart muscle cells that generate contractile force, are lost for good. Yet, in mouse models, the hearts of newborns regenerate readily after injury.
Therefore, this paper proposes a novel cost-sensitive deep neural network (CSDNN)-based mortality prediction model for out-of-hospital acute myocardialinfarction (AMI) patients with hypertension on imbalanced data. Methods The synopsis of our research is as follows.
Despite the detrimental impact of abnormal glucose metabolism on cardiovascular prognosis after myocardialinfarction (MI), diabetes is both underdiagnosed and undertreated. We investigated associations betwee.
Despite this, the patient went on to develop chest pain, which was accompanied by electrocardiographic signs of acute extensive anterior wall myocardialinfarction and elevated troponin I levels.
BackgroundMyocardial fibrosis is a pathological hallmark of heart failure post infarction, emphasizing the need for innovative treatment strategies. Journal of the American Heart Association, Ahead of Print. Mesenchymal stem cellderived EVs, loaded with PAP, were encapsulated in the SA hydrogel (EVsPAP@SA).
Inflammatory diseases impair the reparative properties of endothelial progenitor cells (EPC); however, the involvement of diabetes in EPC dysfunction associated with myocardialinfarction (MI) remains unknown.
The purpose of this study was to investigate the diagnostic value of circ_0013958 in acute myocardialinfarction (AMI) patients and its influence on the prognosis of AMI patients.
Sodium-glucose co-transporter 2 inhibitors (SGLT2i) have been suggested to exert cardioprotective effects in patients with heart failure, possibly by improving the metabolism of ketone bodies in the myocardium.
Previous observational studies have documented an inverse association of birthweight with myocardialinfarction (MI) but a positive association with atrial fibrillation (AF). However, the causality of these as.
High levels of MG53 may attenuate the damage from myocardialinfarction (MI). Furthermore, N6-methyl-adenosine (m6A) methylation is a mode of RNA modification that influences mRNA functions. Whether m6A modifi.
Type 2 diabetes has traditionally been a risk factor for worse prognosis after myocardialinfarction (MI), but major advances have been made in its treatment, and the use of secondary preventive measures has i.
Patients with acute myocardialinfarction (AMI) complicated by cardiogenic shock (CS) face high mortality rates. Extracorporeal Membrane Oxygenation (ECMO) therapy offers critical support in these cases, yet i.
Stress hyperglycemia occurs frequently in patients following acute myocardialinfarction (AMI) and may aggravate myocardial stiffness, but relevant evidence is still lacking. Accordingly, this study aimed to e.
Research design and methods All patients with diabetes and CAD undergoing a coronary angiography between 2010 and 2021 according to the Swedish Angiography and Angioplasty Registry were included. Results Among all patients (n = 38,671), 31% had stable CAD, and 69% suffered an acute myocardialinfarction.
Objective To collate existing qualitative research examining patients’ medication-taking experiences in secondary prevention of acute myocardialinfarction (AMI) and produce new knowledge, a systematic review and meta-synthesis of patient qualitative studies was conducted.
Background Despite restoration of epicardial blood flow in acute ST-elevation myocardialinfarction (STEMI), inadequate microcirculatory perfusion is common and portends a poor prognosis. Further research is needed to optimise the selection of thrombolytic agents and treatment protocols.
The clinical outcomes assessed included all-cause mortality, cardiac-specific mortality, myocardialinfarction (MI), repeat revascularization, stroke, sternal wound complications, and major bleeding. We conducted a comprehensive search across multiple databases for RCTs that directly compared MAG and SAG.
The benefits of complete revascularization over culprit-only coronary artery revascularization were confirmed in older patients with ST-segment elevation myocardialinfarction (STEMI) and multivessel disease over the first 4 years, according to late-breaking research presented in a Hot Line session today at ESC Congress 2024.
A new study by researchers at Karolinska Institutet and Sdersjukhuset shows that advanced ultrasound imaging analyses cannot identify patients who would benefit from beta-blocker treatment after a myocardialinfarction.
Individual outcomes, including all-cause mortality, myocardialinfarction, and revascularization, also showed no significant differences between the two groups. After PS matching, the POCE incidence remained similar between the two groups (3.7% vs. 3.4%, HR 1.01, 95% CI 0.76–1.35, 1.35, p = 0.931).
Percutaneous coronary intervention (PCI) can effectively restore myocardial perfusion in patients with ST-segment elevation myocardialinfarction (STEMI). Nevertheless, STEMI patients may still experience a n.
The purpose of this study was to explore the expression of miR-665 in acute myocardialinfarction (AMI) and evaluate its significance in the diagnosis and prognosis of AMI.
We aimed to develop and validate prediction models for stroke and myocardialinfarction (MI) in pati. Digitalization and big health system data open new avenues for targeted prevention and treatment strategies.
Few studies explored the effect of the combination of glucose sodium-cotransporter-2 inhibitors (SGLT-2i) and glucagon-like peptide-1 receptor agonists (GLP-1RA) on the incidence of cardiovascular events in pa.
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