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Background Contemporary management of spontaneous coronary artery dissection (SCAD) is still controversial. This systematic review of the literature aims to explore outcomes in the patients treated with conservative management vs. invasive strategy. of the patients were diagnosed with non-ST elevated myocardialinfarction (NSTEMI), 36.8%
Background Spontaneous coronary artery dissection (SCAD) and Takotsubo syndrome (TTS) constitute two common causes of nonatherosclerotic acute cardiac syndrome particularly frequent in women. Currently, there is no information comparing long-term clinical outcomes in unselected patients with these conditions. In-hospital events (43.3%
In the absence of these factors it is termed spontaneous coronary artery dissection ( SCAD ). At that time the literature suggested: SCAD was rare , Mostly related to pregnancy , Seen on angiography as a dissection flap , and Managed similarly to MI caused by CAD (ASA, BB, lytics/PCI ). The SCAD cases in Lobo et al. Lobo et al.
This is diagnostic of myocardialinfarction. What is Spontaneous Coronary Artery Dissection (SCAD)? I asked Angie Lobo ( [link] ), a third year intermal medicine resident at Abbott Northwestern Hospital (and Minneapolis Heart Institute) and an aspiring cardiologist, to write a couple paragraphs on SCAD.
Immediate and early percutaneous coronary intervention in very high-risk and high-risk non-ST segment elevation myocardialinfarction patients. Her long term outcome (with very large LAD MI and EF of 30%) is unknown. Currently, SCAD is a diagnosis that can only be established emergently in the cath lab with angiography.
Soviet biologist Trofim Lysenko famously rejected the objective reality of Mendelian genetics because it clashed with the Marxist philosophy that the environment, not genetics, was the primary determinant of outcomes. But what should matter is outcomes not diagnoses. ” What is going on? and 1.7% [ P =0.43]; 12‐month: 0.6%
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