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It should be kept in mind that on occasions, beta-one agonist can result in increased ventricular ectopy e.g., in severe myocardial ischemia (by increasing myocardial demand), or sometimes with congenital long-QT syndrome. Even with tachycardia and a paced QRS duration of ~0.16 J Am Coll Cardiol.
The ECG shows sinus tachycardia with RBBB and LAFB, without clear additional superimposed signs of ischemia. Hopefully a repeat echocardiogram will be performed outpatient. The Initial ECG in Today's Case: As per Dr. Meyers — the initial ECG in today's case shows sinus tachycardia with bifascicular block ( = RBBB/LAHB ).
In terms of ischemia, there is both a signal of subendocardial ischemia (STD max in V5-V6 with reciprocal STE in aVR) AND a signal of transmural infarction of the inferior wall with Q wave and STE in lead III with reciprocal STD in I and aVL. The rhythm is atrial fibrillation. The QRS complex is within normal limits.
Case sent by Magnus Nossen MD, edits by Meyers A previously healthy woman in her 60s presented to an outpatient clinic for palpitations. The ECG there reportedly showed an irregular tachycardia, and the patient was immediately referred to the emergency room. Vitals were within normal limits other than heart rate.
9 However, because troponin is a clear marker of disease severity and a powerful independent predictor of adverse outcomes, it may be quite useful in the ED disposition decision: if troponin is elevated, then outpatient management should be reconsidered. When cTn is elevated, is there a way to differentiate AMI from Non-AMI myocardial injury?
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