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If left untreated, PAD may progress to severe forms known as chronic limb-threatening ischemia (CLTI) and acute limb ischemia (ALI). It has been estimated that less than 5% of patients with PAD in the U.S. are prescribed to participate in a supervised exercise program.”
But it was interpreted as no acute ischemia and the patient was referred to cardiology as Non-STEMI. But clearly this 'Non-STEMI' patient with OMI would have benefited from immediate cath lab activation on arrival, when their first troponin was 11ng/L, rather than after after it rose to 12,000ng/L after 12 hours of refractory ischemia.
Critical limb ischemia (CLTI) is the advanced form of PAD that can result in a lack of healing and limb loss as the most devastating consequence. The team should also include wound nurses, nutritionists, occupational therapists, orthotists, pharmacists, physical therapists, prosthetists, and social workers.
Triage is backed up, and 10 minutes into your shift one of the ED nurses brings your several ECG s that has not been overread by a physician. Remember, in diffuse subendocardial ischemia with widespread ST-depression there may b e ST-E in lead s aVR and V1. The ECG does not show any signs of ischemia. There are also J-waves.
None of these findings are diagnostic of ischemia, but they should give you a high index of suspicion and prompt serial ECGs at a minimum. The patient was diagnosed with esophageal reflux and was being discharged by the nurse when he had a cardiac arrest. Ischemia comes and goes. The formula results in 23.43, just above the 23.4
David Didlake Acute Care Nurse Practitioner Firefighter / Paramedic (ret) @DidlakeDW Expert commentary and peer review by Dr. Steve Smith [link] @smithECGBlog A 57 y/o Female with PMHx HTN, HLD, DM, and current use of tobacco products, presented to the ED with chest discomfort. What’s interesting is that the ECG can only detect ischemia.
Introduction:Tenecteplase (TNK) is becoming the preferred thrombolytic for acute brain ischemia. We subsequently educated constituents including nursing, pharmacy, technical and clinical staff. Stroke, Volume 55, Issue Suppl_1 , Page ATP112-ATP112, February 1, 2024.
David Didlake Firefighter / Paramedic Acute Care Nurse Practitioner @DidlakeDW Peer review and commentary by Dr. Steve Smith [link] @SmithECGblog It is early-summer, approximately 1330 hours, no cloud cover overhead, and 86 degrees with high humidity. There is LBBB-like morphology with persistent patterns of subendocardial ischemia.
David Didlake Firefighter / Paramedic Acute Care Nurse Practitioner @DidlakeDW Peer review provided by Dr. Steve Smith [link] @SmithECGBlog An adult female called 911 for chest discomfort and difficulty breathing. Chapter 6: Introduction to Myocardial Ischemia and Infarction. 2] But there is also Sinus Tachycardia! 2] Birnbaum, Y.,
David Didlake Firefighter / Paramedic Acute Care Nurse Practitioner @DidlakeDW Peer review by Dr. Stephen Smith @smithECGblog I was reviewing ECG’s in our LifeNet database and happened upon this one without any knowledge of clinical circumstances. There is one additional feature – the ST segments of V2-V5 are depressed. ST-elevation, etc.)
David Didlake Acute Care Nurse Practitioner Firefighter / Paramedic @DidlakeDW A 50 y/o Male was taking his dog for a leisurely stroll through the park when he suddenly experienced new onset chest discomfort. He waited for it to subside, but after 30 minutes of persistence he called 911.
One of my most talented readers is a health care assistant (a nursing assistant) who has taken a keen interest in ECGs. Furthermore, there are T-wave changes in V2 and V3 which are highly suggestive of ischemia, but difficult to localize: anterior? And they teach me a lot. He can beat nearly anyone. You don't have to be a genius.
Our triage nurse therefore ordered an ECG for him (which is standard in our dept for epigastric pain patients): What do you think? They cannot be assumed due to LV strain ( and they cannot be assumed to represent ischemia ). He denied chest pain of any sort and his vitals were all normal.
In spite of aggressive questioning, he denied chest pain, but he did tell one triage nurse that he had had some chest burning, and so he underwent an ECG: There are deep Q-waves and QS-waves in precordial leads V2-V3, with a bit of R-wave left in V4. This 42 yo diabetic male presented with cough and foot pain. This was recorded 2.5
David Didlake Firefighter / Paramedic Acute Care Nurse Practitioner @DidlakeDW Peer review provided by Dr. Steve Smith @SmithECGblog I was conducting QA/QI on two very recent cases and was struck by the uniqueness of both. It’s important to stress the presence of a normal QRS (i.e.,
It had started just after nursing her newborn, about an hour prior, and she described it as a severe non-pleuritic “pressure” radiating to the back. This strongly suggests reperfusing RCA ischemia. This is written by Brooks Walsh. link] A 30 year-old woman was brought to the ED with chest pain. There is also a Q-wave in III.
David Didlake Firefighter / Paramedic Acute Care Nurse Practitioner @DidlakeDW Peer review provided by Dr. Steve Smith [link] @SmithECGblog A 72 y/o Male experiences a syncopal episode while seated. Lead V2 shows RR’ QRS configuration, and although ST depression is otherwise expected here, the discordance is a bit excessive.
If there is polymorphic VT with a long QT on the baseline ECG, then generally we call that Torsades, but Non-Torsades Polymorphic VT can result from ischemia alone. It would be difficult to get a nurse to give it faster! If there is a pulse, you would call it Torsades. However it is classified is not so important! Is 40 mEq too much?
It’s an intubated septic nursing home patient." I received this ECG in a text message, with the message: "Hey, these look like hyperacute T waves to me, what do you think? Here is her old ECG:" What do you think? Here is my response: "There is something wrong with this ECG. It might be another case of pulse tapping artifact.
In the evening, a middle-aged man complained of chest pain at the nursing home. Nurses found him with a BP of 50/30 and heart rate of 130 and called EMS. He was awake, with a pulse of 130 and BP of 50/30. Fluids were started. The patient arrived alert but cool and clammy. His chest pain was vague. He complained of chronic dyspnea.
David Didlake Firefighter / Paramedic Acute Care Nurse Practitioner @DidlakeDW Expert commentary provided by Dr. Ken Grauer CASE 1 An 82 y/o Male called 911 for sudden onset dizziness while at rest. Upon arrival he was found alert and oriented, and without gross distress. He denied difficulty breathing, epigastric pain, or chest discomfort.
This case reminds me of this 27 year old totally healthy nurse who was previously healthy, presented with acute pulmonary edema and the below ECG that is diagnostic of proximal LAD occlusion, and was dismissed because of her age. This gets drilled into them.
The nurse alerted the MD because the patient was still symptomatic, diaphoretic and “looking unwell”. Even though they were passed the 12 hour mark traditionally associated with reperfusion benefits, ongoing ischemia requires emergent angiogram On assessment, the patient appeared uncomfortable, leaning forward in his chair. Shroff, G.
His triage EKG is shown below: There is left bundle branch block, so the EKG must be evaluated for ischemia by Smith-modified Sgarbossa criteria. There is evidence of transmural ischemia of the posterior wall as well. Leads V1 to V4 have down-up shaped T waves typical of ischemia and atypical of LBBB.
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