Remove Heart Failure Remove Ischemia Remove Pacemaker
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Torsade in a patient with left bundle branch block: is there a long QT? (And: Left Bundle Pacing).

Dr. Smith's ECG Blog

Place temporary pacemaker 3. It should be kept in mind that on occasions, beta-one agonist can result in increased ventricular ectopy e.g., in severe myocardial ischemia (by increasing myocardial demand), or sometimes with congenital long-QT syndrome. See this post: How a pause can cause cardiac arrest 2.

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A fascinating electrophysiology case. What is this wide complex tachycardia, and how best to manage it?

Dr. Smith's ECG Blog

She had a single chamber ICD/Pacemaker implanted several years prior due to ventricular tachycardia. Are you confident there is no ischemia? The heart rate is about 130 bpm. The heart rate could be compatible with that of a 2:1 conducted atrial flutter. The last echocardiography 12 months ago showed HFmrEF.

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Physical Examination as a Helpful Aid in Decision-Making in Challenging ECGs

Dr. Smith's ECG Blog

His medical history includes hypertension, a decade-long battle with diabetes, ischemic heart disease, a coronary bypass graft surgery ten years ago, a diagnosis of congestive heart failure for the last five years, and a prior ICD implantation five years ago. Thus VT is very probable.

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A 50-something with chest pain. Is there OMI? And what is the rhythm?

Dr. Smith's ECG Blog

The fact that R waves 2 through 6 are junctional does make ischemia more difficult to interpret -- but not impossible. The Queen of Hearts does not care about rhythm analysis, she simply looks at the ECG and decides whether it represents OMI or not. second that sinus-conducted beats #1,7,8 tell us is needed for normal AV conduction ).

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This middle-aged patient presented with SOB, weakness, and mild pulmonary edema.

Dr. Smith's ECG Blog

There was no evidence of ischemia. She had a permanent pacemaker implanted. After pacer AND conversion to sinus rhythm: Computer diagnosis: IMPRESSION ELECTRONIC VENTRICULAR PACEMAKER ABNORMAL RHYTHM ECG What is missing from this interpretation? We are not told how ischemia has been ruled out in this case. Hyperkalemia.

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Syncope and Block

EMS 12-Lead

Such findings would normally suggest primary ischemia with concomitant surveillance of coronary occlusion, but these ST/T changes might very well be secondary to the Escape mechanism at hand. The patient care narrative states no further changes in heart rate with persistent LBBB morphology. European Heart Journal, 28 , 2449-2455. [7]

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American College of Cardiology (ACC24) Show Preview: Advancing Cardiovascular Care for All

DAIC

Rapid Fire Challenging Structural Heart Imaging Cases with Heart Team Panel; Follow-up of Pacemakers and ICDS for the Non-electrophysiologist; The Real Reasons Your Patient with Heart Failure was Readmitted: Noncardiac Comorbidities, Geriatric Syndromes and Social Determinants of Health; and Death by a Thousand Cuts!