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Exercise catheterization in adults post?Fontan with normal and abnormal hemodynamic criteria: insights into normal Fontan physiology

European Journal of Heart Failure

Moreover, the definitions of elevated exercise pulmonary artery (PA) and PA wedge pressure (PAWP) for this population have not been described. There was no difference in exercise arterial O 2 saturation (87% [81;92] vs 89% [85;93], pā€‰=ā€‰0.29), while exercise PA pressure (27 [23;31] vs 16 [14.5;19.5]

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Physiology Friday #205: What Limits VO2 Max? It Might Depend on Your Fitness Level.

Physiologically Speaking

Welcome to the Physiology Friday newsletter. Physiologically Speaking is a reader-supported publication. There’s a long-running debate in exercise physiology about what limits VO2 max. However, after 8 weeks of exercise training, the limitation of VO2 max shifts to oxygen transport.

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Is the Heart Really a Pump? New Perspectives on Cardiovascular Physiology

Physiologically Speaking

Cardiovascular physiology has long held that the heart is a mechanical pump and that the heart’s propulsive power is the main driver of blood flow throughout the body. However, the cardiocentric view of circulation fails to explain blood flow regulation during exercise and other unique scenarios.

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A young woman with palpitations. What med is she on? With what medication is she non-compliant? What management?

Dr. Smith's ECG Blog

Before continuing flecainide, he had me get on a treadmill at full dose and at full exercise (18 minutes) and measured the QRS to be certain that the QRS did not lengthen at all. It simply does not make physiologic sense to suddenly see an all-negative QRS complex in this most lateral chest lead. Coincidence?).