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Case Report: Loeffler endocarditis as a cause of left ventricular thrombosis in young women: a case study and literature review

Frontiers in Cardiovascular Medicine

Left ventricular thrombus formation and cardiac insufficiency were evident on echocardiogram, while multiple acute or subacute cerebral infarctions were visible on brain magnetic resonance imaging. Here, we report a rare case of a 28-year-old female patient who presented with chest tightness and dizziness.

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Abnormal echocardiographic finding mimicking paracardiac cystic lesion

Heart BMJ

Previous medical interventions included a spectrum of procedures, including catheter-directed thrombectomy for popliteal artery aneurysms with thrombosis, vascular bypass grafting for cerebral-anterior communicating artery aneurysms and arch replacement and stent implantation for aortic dissecting aneurysms.

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Combined left atrial appendage occlusion and catheter ablation procedure for left atrial arrhythmias: A real‐world, propensity‐matched analysis

Journal of Cardiovascular Electrophysiology

vs. 30.4%, p =0.07) and device related thrombosis (4.5% vs. 4.5%, p =0.96) on transesophageal echocardiogram did not differ. Both major (1.4% vs. 2.1%, p =0.72) and minor (27.8% vs. 19.4%, p =0.17) in-hospital complications were similar between the combined and control group, respectively.

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Why we need continuous 12-lead ST segment monitoring in Wellens' syndrome

Dr. Smith's ECG Blog

It is important to recognize that coronary thrombosis is dynamic , with spontaneous opening and lysing of the thrombus in the infarct-related artery (we all have endogenous tPA and plasmin to lyse thrombi). There are those who think that an echo that is done after resolution of ischemia is sensitive for that previous ischemia.

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A Child with Blunt Trauma

Dr. Smith's ECG Blog

Blunt cardiac injury my result in : 1) Acute myocardial rupture with tamponade 2) Valve rupture (tricuspid, aortic, mitral) 3) Coronary thrombosis or dissection (and thus Acute MI) from direct coronary blunt injury 4) Dysrhythmias of all kinds. She was discharged to home feeling just fine.

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Upon arrival to the emergency department, a senior emergency physician looked at the ECG and said "Nothing too exciting."

Dr. Smith's ECG Blog

Here is the cath report: Echocardiogram: There is severe hypokinesis of entire LV apex and apical segment of all the walls. MINOCA may be due to: coronary spasm, coronary microvascular dysfunction, plaque disruption, spontaneous coronary thrombosis/emboli , and coronary dissection. ng/mL by 4th generation and older assays.)

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Concerning EKG with a Non-obstructive angiogram. What happened?

Dr. Smith's ECG Blog

The commonest causes of MINOCA include: atherosclerotic causes such as plaque rupture or erosion with spontaneous thrombolysis, and non-atherosclerotic causes such as coronary vasospasm (sometimes called variant angina or Prinzmetal's angina), coronary embolism or thrombosis, possibly microvascular dysfunction.

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