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Now it is a full blown STEMI of 3 myocardial territories: inferior, posterior, and lateral But at least it does not call it "Normal." Formal echocardiogram: Systolic function is at the lower limits of normal. Learning Points: You cannot trust conventional algorithms even to find STEMI(+) OMI, even when they say "normal ECG."
Precordial ST depression may be subendocardial ischemia or posterior STEMI. If you thought it might be a posterior STEMI, then you might have ordered a posterior ECG [change leads V4-V6 around to the back (V7-V9)]. So there was 3-vessel disease, but with an acute posterior STEMI. There is no ST elevation. See the list below.
This has been termed a “STEMI equivalent” and included in STEMI guidelines, suggesting this patient should receive dual anti-platelets, heparin and immediate cath lab activation–or thrombolysis in centres where cath lab is not available. See this case: what do you think the echocardiogram shows in this case?
The last section is a detailed discussion of the research on aVR in both STEMI and NonSTEMI. The additional ST Elevation in V1 is not usually seen with diffuse subendocardial ischemia, and suggests that something else, like STEMI from LAD occlusion, could be present. Updates on the Electrocardiogram in Acute Coronary Syndromes.
An initial electrocardiogram (ECG) is provided below. A rapid echocardiogram was performed, revealing an ejection fraction of 20% with thinning of the anterior-apical walls. His current medication regimen includes apixaban, carvedilol, perindopril, spironolactone, torasemide, dapagliflozin, amiodarone, and ivabradine.
You've read in my previous posts that I have a lot of evidence that Wellens' represents spontaneously reperfused STEMI in which the STEMI went unrecorded. New ST elevation diagnostic of STEMI [equation value = 25.3 This T-wave inversion morphology is very specific for Wellens' waves. Computerized QTc = 417. Patel DJ, et al.
Formal Echocardiogram: The estimated left ventricular ejection fraction is 58 %. Emergent cardiac outcomes in patients with normal electrocardiograms in the emergency department. These include about 60 occlusion MI (OMI) with clear ST segment elevation (none of which would be called “Normal” by the computer) and about 165 Non-STEMI.
You will note that it is essentially an unremarkable electrocardiogram except for some PACS. Unfortunately there is no echocardiogram accessible because the patient checked himself out of the hospital in order to get back to his home state before it could be completed. In the available view, the RCA appears fully occluded.
They recorded a prehospital ECG and diagnosed STEMI and activated the cath lab prehospital. Next day, a stress echo was done: The exercise stress echocardiogram is normal. The stress electrocardiogram is non-diagnostic. I heard that a prehospital cath lab activation was on its way. No wall motion abnormality at rest.
Smith , d and Muzaffer Değertekin a DIFOCCULT: DIagnostic accuracy oF electrocardiogram for acute coronary OCClUsion resuLTing in myocardial infarction. He visited an outpatient clinic for it and an echocardiogram and exercise stress test was normal. His first electrocardiogram ( ECG) is given below: --Sinus bradycardia.
A prior ECG from 1 month ago was available: The presentation ECG was interpreted as STEMI and the patient was transferred emergently to the nearest PCI center. Formal echocardiogram showed normal EF, no wall motion abnormalities, no pericardial effusion. Induced Brugada-type electrocardiogram, a sign for imminent malignant arrhythmias.
2) The STE in V1 and V2 has an R'-wave and downsloping ST segments, very atypical for STEMI. Cardiology was consulted and they agreed that the EKG had an atypical morphology for STEMI and did not activate the cath lab. Induced Brugada-type electrocardiogram, a sign for imminent malignant arrhythmias. Bicarb 20, Lactate 4.2,
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