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The ECGs show a wide complex, irregularly irregular tachycardia. The therapeutic and diagnostic cardiac electrophysiological uses of adenosine We can again see shortening of the PR interval and widening of the QRS complex with delta waves. On arrival to the ED, he was noted to be in a wide complex tachycardia with a rate in the 240s.
Here is his 12-lead: There is a wide complex tachycardia with a rate of 257, with RBBB and LPFB (right axis deviation) morphology. Read about Fascicular VT here: Idiopathic Ventricular Tachycardias for the EM Physician Case Continued He was completely stable, so adenosine was administered. See Learning point 1 below. Arch Intern Med.
See here for management of Polymorphic Ventricular Tachycardia , which includes Torsades. Could the dysrhythmias have been prevented? Severe hypokalemia in the setting of STEMI or dysrhythmias is life-threatening and needs very rapid treatment. Learning Points: 1. 5-10 mEq over 5-10 minutes is appropriate for a K of 1.8
Otherwise vitals after intubation were only notable for tachycardia. An initial EKG was obtained: Computer read: sinus tachycardia, early acute anterior infarct. She has not yet been seen by electrophysiology or had further genetic testing for Brugada syndrome. A rectal temperature was obtained which read 107.9
Here was his ED ECG: There is sinus tachycardia (rate about 114) with nonspecific ST-T abnormalities. An ECG was recorded: This shows a regular narrow complex tachycardia at a rate of about 160. See my quick review of atrial tachycardia below) The tachycardia spontaneously resolved. BP:143/99, Pulse 109, Temp 37.2 °C
If the patient has Abnormal Vital Signs (fever, hypotension, tachycardia, or tachypnea, or hypoxemia), then these are the primary issue to address, as there is ongoing pathology which must be identified. Electrophysiologic studies were performed in selected patients only as clinically appropriate.
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