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ECG Blog #435 — Did Cath Show Acute Ischemia?

Ken Grauer, MD

That said — the ECG in Figure-1 should prompt the following considerations: The symmetric chest lead T wave inversion in ECG #1 could be a sign of coronary disease, potentially with acute ischemia. During my decades of working with residents when hospital Attending — by far, the most commonly overlooked vital sign was respiratory rate.

Blog 171
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Dynamic OMI ECG. Negative trops and negative angiogram does not rule out coronary ischemia or ACS.

Dr. Smith's ECG Blog

This confirms that the pain was ischemia and is now resovled. We documented that the majority of stenotic lesions had compensatory enlargement and thus exhibited remodeling. The cardiology fellow agreed with plan for emergent cath and escorted the patient to the cath lab. The i nitial hs troponin I returned 75%.

Ischemia 121
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New in Clinical Documents | Management of Lower Extremity PAD Focus of New ACC/AHA Guideline

American College of Cardiology

The new 2024 Guideline on the Management of Lower Extremity Peripheral Artery Disease (PAD) released by the ACC and the American Heart Association (AHA) offers the latest, evidence-based recommendations to guide clinicians in the diagnosis and treatment of lower extremity PAD across its four clinical presentation subsets: asymptomatic disease, chronic (..)

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A Placebo-Controlled Trial of Percutaneous Coronary Intervention for Stable Angina

The New England Journal of Medicine

In a randomized trial of PCI in patients with stable angina who were receiving little or no antianginal medication and had documented ischemia, PCI resulted in a better health status with respect to angina than placebo at 12 weeks.

Angina 40
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A fascinating electrophysiology case. What is this wide complex tachycardia, and how best to manage it?

Dr. Smith's ECG Blog

Are you confident there is no ischemia? Primary VT , and the VT with tachycardia is causing ischemia with chest discomfort (supply-demand mismatch/type 2 MI)? Ischemia from ACS causing the chest discomfort, with VT another consequence (or coincidence)? Do you agree with this strategy? How can you better assess the ST segments?

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Ischemic ST depression maximal in V1-V4 (vs. V5-V6), even if less than 0.1 millivolt, is specific for Occlusion Myocardial Infarction (vs. subendocardial non-occlusive ischemia)

Dr. Smith's ECG Blog

If this STD were due to LVH or to subendocardial ischemia, rather than posterior OMI, it would be maximal in V5 and V6. If I saw this without the STD V2-V4, I would not make anything of it, and even with that precordial STD, I am not convinced that it is a manifestation of ischemia. Alcohol intoxication? V5-V6), even if less than 0.1

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An elderly male with acute altered mental status and huge ST Elevation

Dr. Smith's ECG Blog

or basilar ischemia. The providers documented concern for ST elevation in the precordial and lateral leads as well as a concern for hyperkalemic T waves in the setting of succinylcholine administration. Preliminary findings documented in the cath lab were “Anterior STEMI and no significant coronary artery disease.” (!!!)

STEMI 114