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There were no dysrhythmias on cardiac monitor during observation. This discussion comes from this previous post: Hyperthermia and ST Elevation Discussion Brugada Type 1 ECG changes are associated with sudden cardiac death (SCD) and the occurrence of ventricular dysrhythmias. He was found to be influenza positive. Is there fever again?
If you don't know what the dysrhythmia is, then try procainamide. As I discussed and documented in Lesson 1 of My Comment at the bottom of the page in the April 2, 2022 post of Dr. Smith's ECG Blog — certain patients may remain in sustained VT not only for hours — but even for days! What to do now? So I would give procainamide.
I’ve attached an article and an abstract (that article is in Japanese unfortunately … ) that do document that you CAN however on occasion find AIVR in otherwise healthy children — and I suppose that IS what we have here. In ECG #3 , the low atrial rhythm rate becomes slight faster than the AIVR rhythm — so that’s why it again takes over.
So the real QT is shorter, but the computer does not mention the U-wave, and the U-wave is as important as the T-wave in predicting cardiac dysrhythmias. Document in the patient's chart that rapid infusion is intentional in response to life-threatening hypokalemia." This is an extremely dangerous ECG. The K returned at 1.9
Could the dysrhythmias have been prevented? Document in the patient's chart that rapid infusion is intentional in response to life-threatening hypokalemia." Severe hypokalemia in the setting of STEMI or dysrhythmias is life-threatening and needs very rapid treatment. If cardiac arrest from hypokalemia is imminent (i.e.,
The limb lead abnormalities appear to be part of the Brugada pattern, as described in this article: Inferior and Lateral Electrocardiographic RepolarizationAbnormalities in Brugada Syndrome Discussion Brugada Type 1 ECG changes are associated with sudden cardiac death (SCD) and the occurrence of ventricular dysrhythmias.
Document in the patient's chart that rapid infusion is intentional in response to life-threatening hypokalemia." I could find very little literature on the treatment of severe life-threatening hypokalemia. There is particularly little on how to treat when the K is less than 2, and/or in the presence of acute MI. "If
Inferior MI results in scar tissue which is a likely source of a re-entrant ventricular dysrhythmia. As always — it’s nice when we have “the Answer” , here in the form of an EP study documenting the absence of any SVT — with confirmation that the rhythm is VT. This would be the likely source of the VT.
See below how this has been documented. Atrial dysrhythmias, and atrial fi brillation in particular, are frequently misdiagnosed by computer algorithms and then by the physician who overreads them. Shah and Rubin studied the computer rhythm interpretation of 2160 12-lead ECGs, compared to 2 cardiologists [ 18 ].
Thus, if there is documented sinus bradycardia, and no suspicion of high grade AV block, at the time of the syncope, this is very useful. Dysrhythmia, pacer), 4) valvular heart disease, 5) FHx sudden death, 6) volume depletion, 7) persistent abnormal vitals, 8) primary CNS event __ 3) Mendu ML et al.
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