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A 50-year-old woman with dextrocardia and D-transposition of the great arteries presented with a “RV defib lead impedance” remote-monitoring alert from her implantable cardioverter defibrillator (ICD). The superior limb of the baffle was stenosed and required stenting. An epicardial pacemaker was implanted.
While in the hospital, he had witnessed ventricular fibrillation (VF) arrest for which he received external defibrillation. He had minimal in-stent restenosis on angiography but had only minimal cardiac enzyme elevation and did not have antecedent chest pain before either of his events.
VF was refractory to amiodarone, lidocaine, double-sequential defibrillation, esmolol, etc. He has a h/o of 3 vessel disease and stents and his pain has been on and off for days. Then the patient would have been taken to the critical care area with a defibrillator at his side while waiting for the cath lab to be ready.
She was defibrillated successfully from ventricular fibrillation and developed a perfusing rhythm. Pressors were required, and the patient was transported to the cath lab with a door to balloon time of 60 minutes, where a proximal dominant RCA occlusion was opened and stented. She was intubated.
The patient had 2 ventricular fibrillation arrests during transport, but was immediately defibrillated both times, and was awake in the ED, when the following ECG was recorded: The ST elevation has mostly resolved on this ECG, and were it not for the arrest and the prehospital ECG, this would not be a slam dunk diagnosis.
We describe a case of ventricular pacemaker spikes delivered on the T wave causing PMVT.Case:A 53-year-old female with CAD s/p stent, postpartum cardiomyopathy s/p Bi-V CRT-D (Boston Scientific G124), and paroxysmal atrial fibrillation presented for elective endoscopy and colonoscopy to evaluate her dysphagia and abdominal pain.
February 2024 FDA Approvals: Innovations in Cardiovascular Interventions XACT Carotid Stent System (Approved: 02/07/2024) This approval expands the indications to be used during a Transcarotid Artery Revascularization (TCAR) procedure to prevent future strokes.
She was found to be in ventricular fibrillation and was defibrillated 8 times without a single, even transient, conversion out of fibrillation. She was immediately intubated during continued compressions, then underwent a 9th defibrillation, which resulted in an organized rhythm at 42 minutes after initial arrest. It was stented.
We offer a wide range of diagnostic and treatment services including: Coronary artery bypass surgery Angioplasty and stenting Heart valve surgery Pacemaker and defibrillator implantation Cardiac rehabilitation We believe every patient deserves personalized care.
After the second defibrillation the patient had an organized rhythm: Bradycardic escape/agonal rhythm, with large ST deviations. The patient was taken back to the cath lab, where 100% proximal in-stent rethrombosis was found and treated. It should have been shocked at least 10 seconds ago. This is diagnostic of re-occlusion.
He denied any known medical history, specifically: coronary artery disease, hypertension, dyslipidemia, diabetes, heart failure, myocardial infarction, or any prior PCI/stent. Despite immediate chest compressions, and multiple rounds of defibrillation, he could not be resuscitated. No appreciable skin pallor.
During angiogram in the cath lab, the patient suffered two episodes of ventricular fibrillation for which he was successfully defibrillated. Two stents were placed with resultant TIMI 3 flow.
He was defibrillated immediately and had return of normal mental status. They found an acute lesion of the LAD at the site of the prior stents, including 70% proximal LAD lesion and 95% mid-LAD stenosis with TIMI 3 flow at the time of cath. The LAD lesion was acute and required 3 stents to restore flow.
100% proximal LAD successfully stented. Defibrillated out of v fib in the cath lab. "Bedside echo revealed anteroseptal wall motion abnormality at which point I activated a code STEMI. Cardiology agreed to take the pt to the lab but thought it would likely be negative. Initial TnI was negative. "I
Interventions that may prolong life include implantation of a defibrillator. Patients with a very weak hearts may be prone to life threatening heart rhythm disturbances and therefore a defibrillator within the chest can detect this and deliver a life-saving shock there and then and thereby prolong life.
At cath, he immediately had incessant Torsades de Pointes requiring defibrillation 7 times and requiring placement of a transvenous pacer for overdrive pacing at a rate of 80. This was stented. The patient was intubated, given antiplatelet and antithrombotic therapy, 10 mEq of KCl IV was started, and sent to the cath lab.
These issues can only be addressed in an ICCU (Intensive Coronary Care Unit) setting, where temporary pacemakers and defibrillators are available. Then angioplasty is performed, and a medical device called a stent (metallic scaffold) is deployed in the artery to open the blood flow. This is known as a pharmaco-invasive approach.
After stent deployment, we often see improvement in the ST-T within seconds or minutes. Here is the final angiogram following placement of a stent in the ostial RCA. 2:04 PM, post stent deployment You can see that even after complete restoration of flow, the ECG still looks terrible, V most of all. SanzRuiz, R., Solis, J., &
In this case, you should get a second defibrillator and perform double sequential external defibrillation (DSED). Simply attach a second defibrillator as shown in the diagram below and deliver max shocks from both devices simultaneously. In the second case, the patient never converted meaning the shock did not do its job at all.
Submitted anonymously, written by Willy Frick A man in his 70s with a history of remote MI (details unavailable) and prior stent placement presented to cardiology clinic for routine follow up. He complained of days to weeks of palpitations and dyspnea. His clinic ECG is shown. What do you think? For How Long can a Patient remain in Stable VT?
Defibrillation was performed, and ROSC was achieved. Total proximal LAD occlusion was found and stented at angiography soon after the ECG above. Unfortunately, the ECG was interpreted as no significant change from prior , "no STEMI"!! He was sent back to the waiting room, where he suffered a VF arrest.
The report describes heavy plaque in the proximal RCA by IVUS, but no lesions in the previously occluded RPL branch and no stent was deployed. Throughout this process, the patient had repeated VF and was defibrillated 8 times. It is consistent with an inferior LV aneurysm. Prolonged thrombectomy effort was unsuccessful.
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