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Chronic Pulmonary Disease Lung diseases like chronic obstructive pulmonary disease (COPD) can lead to pulmonary hypertension, which in turn can cause the right side of the heart to enlarge, a condition known as cor pulmonale. Implantable Cardioverter-Defibrillator (ICD) to help manage dangerous heart rhythms.
He was defibrillated, but they also noticed that he was being internally defibrillated and then found that he had an implantable ICD. He was unidentified and there were no records available After 7 shocks, he was successfully defibrillated and brought to the ED. There was no bystander CPR. l/min cardiac output.
She was unable to be defibrillated but was cannulated and placed on ECMO in our Emergency Department (ECLS - extracorporeal life support). After good ECMO flow was established, she was successfully defibrillated. Here is a case of ECMO defibrillation with near shark fin that was due to proximal LAD occlusion. The K was normal.
Background:Epicardial patch defibrillators (EPDs) were commonly implanted in the 1990s for secondary prevention of sudden cardiac death. Circulation, Volume 150, Issue Suppl_1 , Page A4139677-A4139677, November 12, 2024. Despite being largely obsolete, some EPDs remain in patients and can cause late complications.
It cannot be treated with a defibrillator and often leads to death. Ventricular fibrillation is a type of irregular heartbeat that can cause the heart to stop beating, but an electric shock from a defibrillator can trigger the beating again.
The receiving staff suspects pulmonary embolism due to S1Q3T3 on the ECG and administers TPA. The patient did have massive pulmonary emboli, but he also had profound intraventricular and subarachnoid hemorrhages. On ED arrival ROSC is achieved. The patient was ultimately discharged with a poor neurologic outcome.
CT of the chest showed no pulmonary embolism but bibasilar infiltrates. Because she has cardiomyopathy and ventricular dysrhythmias, the pacer included an Implanted Cardioverter-Defibrillator (ICD) Echo 6 days later after CRT: Normal estimated left ventricular ejection fraction. She was intubated. No wall motion abnormality.
Effects of sacubitril/valsartan with or without background beta-blocker therapy in patients with heart failure with reduced ejection fraction from PARADIGM-HF.
Over the past years, patients with tetralogy of Fallot (TOF) have experienced numerous improvements in different treatment modalities consisting of a mixture of surgical procedures, transcatheter interventions including ablation therapy, and implantable cardioverter-defibrillator implantation procedures.
Join us as we examine the landmark approvals that are revolutionizing patient outcomes. Clinical Trial Results: Aprocitentan reduced and maintained lower BP levels over time, both in SiSBP and SiDBP. BP-lowering effects were consistent across different demographic and clinical subgroups.
She was successfully revived after several rounds of ACLS including defibrillation and amiodarone. Conduction and refractoriness alternans may be seen with WPW-related as well as AV Nodal-dependent reentr y tachycardias — atrial fibrillation — acute pulmonary embolus — myocardial contusion — and severe LV dysfunction.
Today's case reminds us of the intuitive logic that if a patient has a shockable arrest ( ie, VFib ) — and following successful defibrillation shows evidence of acute OMI ( even if STEMI criteria are not necessarily fulfilled ) — that such patients have much to gain from immediate cath with PCI. (
It was reportedly a PEA arrest; there was no recorded V Fib and no defibrillation. The morphology of V2-V4 is very specific in my experience for acute right heart strain (which has many potential etiologies, but none more common and important in EM than acute pulmonary embolism). CT angiogram showed extensive saddle pulmonary embolism.
Oral anticoagulation also reduced a composite of cardiovascular death, all-cause stroke, peripheral arterial embolism, myocardial infarction or pulmonary embolism (RR 0.85, 95% CI 0.73-1.00, We used random-effects models for meta-analysis and rated the quality of evidence using the GRADE framework. 1.00, I2=0%; moderate-quality evidence).
Thrombus can sometimes occur when there is a central venous catheter or a multiple pacemaker or defibrillator leads there that can cause thrombus formation. Right atrial hypertrophy as in tricuspid stenosis, pulmonary stenosis and pulmonary hypertension. But in a VSD with pulmonary hypertension A wave is not prominent.
CASTLE-AF randomized 363 patients with atrial fibrillation and left ventricular ejection fraction of 35% or less, NYHA class II-IV heart failure and having an implanted defibrillator to either catheter ablation or medical therapy with rate or rhythm control [5]. Among the subjects of CABANA trial 35% had NYHA class above II at baseline.
Bedside ultrasound showed no effusion and moderately decreased LV function, with B-lines of pulmonary edema. At cath, he immediately had incessant Torsades de Pointes requiring defibrillation 7 times and requiring placement of a transvenous pacer for overdrive pacing at a rate of 80. He appeared to be in shock.
These issues can only be addressed in an ICCU (Intensive Coronary Care Unit) setting, where temporary pacemakers and defibrillators are available. Therefore, seeking early admission to the ICCU is the initial step toward addressing such problems.
she had severe pulmonary edema. This is the etiology of the blood from her nose and mouth (frothy bloody pulmonary edema) This is what frothy bloody pulmonary edema looks like. Subarachnoid hemorrhage causes extreme central catecholamine output, resulting in stress cardiomyopathy, just like takotsubo. From this site.
Second , when you have a rhythm problem, you are likely to be able to fix the problem with electricity (cardioversion, defibrillation, pacing). Clinically this means that as long as the patient remains hemodynamically stable We can continue with treatment of her acute pulmonary condition. What happens in ECG #3 ?
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