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This usually represents posterior OMI, but in tachycardia and especially after cardiac arrest, this could simply be demand ischemia, residual subendocardial ischemia due to the low flow state of the cardiac arrest. This prompted cath lab activation. On arrival to the ED, this ECG was recorded: What do you think?
BackgroundCatecholaminergic polymorphic ventricular tachycardia (CPVT) is a rare inherited arrhythmia disorder characterized by ventricular arrhythmia triggered by adrenergic stimulation.Case presentationA 9-year-old boy presented with convulsions following physical exertion. Genetic testing revealed a pathogenic variant of RYR2:c.720G>A
The main secondary study endpoints are all-cause mortality, cardiovascular mortality, incidence of implantable cardioverter-defibrillator (ICD) therapy, hospitalizations, quality of life, time to first ICD therapy, number of device-detected ventricular tachycardia/ventricular fibrillation episodes, left ventricular function, and exercise tolerance.
The abnormal heart rhythms can further lead to death because of ventricular tachycardia and ventricular fibrillation. These issues can only be addressed in an ICCU (Intensive Coronary Care Unit) setting, where temporary pacemakers and defibrillators are available. Get some exercise regularly. Manage diabetes.
Previously healthy, taking no medication and exercising regularly. No anginal symptoms asymptomatic during physical exercise. During observation in the ED the patient had multiple self-terminating runs of Non-Sustained monomorphic Ventricular Tachycardia (NSVT). Below in Figure-1 is this patient's admission ECG.
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