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Despite otherwise normal vital signs, she was appropriately triaged to the criticalcare area of the ED. She was awake, alert, well perfused, with normal mental status and overall unremarkable physical exam except for a regular tachycardia, possible rales at both bases, some mild RUQ abdominal tenderness. What is the Diagnosis?
He was rushed by residents into our criticalcare room with a diagnosis of STEMI, and they handed me this ECG: There is sinus tachycardia with ST elevation in II, III, and aVF, as well as V4-V6. ACS and STEMI generally do not cause tachycardia unless there is cardiogenic shock. He had this ECG recorded.
His previous echo one month prior shows the same thing: “consistent with old infarct in LAD vascular territory, with EF 45%” "I think there is something else causing his tachycardia which is exaggerating his EKG findings and mimicking an acute myocardial infarction." The patient spontaneously converted back to sinus tachycardia.
There is a regular wide complex tachycardia. I brought the patient to the criticalcare area and told the providers I thought it was atrial flutter with 2:1 AV conduction, but there is an outside chance that it is VT. Remember : Adenosine is safe in Regular Wide Complex Tachycardia. If it is VT, there will be no effect.
Colin is an emergency medicine resident beginning his criticalcare fellowship in the summer with a strong interest in the role of ECG in criticalcare and OMI. We can see enough to make out that the rhythm is sinus tachycardia. Written by Colin Jenkins. Edits by Willy Frick.
See here for management of Polymorphic Ventricular Tachycardia , which includes Torsades. Crit Care Med. 1991 May;19(5):694-9 Objective: To evaluate the efficacy and safety of potassium replacement infusions in critically ill patients. Setting: Multidisciplinary criticalcare unit. Design: Prospective cohort study.
After initiating treatment for hyperkalemia, repeat ECG showed resolution of Brugada pattern: The ECG shows sinus tachycardia. It is critically important for all EM and criticalcare providers to have an intimate understanding of hyperkalemia and its ECG findings. A Very Wide Complex Tachycardia. Use Lewis Leads!!
While calling for some help and arranging to have her transported to our criticalcare zone, I got this quick ultrasound which confirmed my suspicion: This quick view was all I was able to obtain in the circumstances. One looks for sinus tachycardia and diffuse low voltage but many conditions produce these nonspecific findings.
Although the shock is no doubt partly a result of poor pump function, with low stroke volume, especially of the RV, it should be compensated for by tachycardia. The patient arrived at the Emergency Dept criticalcare area and had this ECG recorded: The sinus bradycardia persists. This is a perfect indication for atropine.
She had this ECG recorded: Obvious massive anterior STEMI She was quickly brought to the criticalcare area and the cath lab was activated. The blood pressure was 170/100 in the criticalcare area. She has no SOB and no prior medical history. Her initial BP was 203/124.
Multidisciplinary criticalcare management of electrical storm. This was overtaken by a predominance of sympathetic surge ( tachycardia, persistent ST elevation development of electrical "storm" with failure to respond to recurrent defibrillation ). [link] Jentzer, J. Noseworthy, P. Kashou, A. Chrispin, J., Tisdale, J.
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