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He was rushed by residents into our criticalcare room with a diagnosis of STEMI, and they handed me this ECG: There is sinus tachycardia with ST elevation in II, III, and aVF, as well as V4-V6. At first glance, it seems the patient is having a STEMI. Then ACS (STEMI) might be primary; this might be cardiogenic shock.
I took the patient to the criticalcare area and questioned him more on the way. Another ECG was recorded while awaiting the cath team: Now there is STEMI Let's look at that first (prehospital ECG) again: Very subtle! The pain had been intermittent until an hour before arrival, when he called 911. We activated the cath lab.
It was tested on a large database of known outcomes and was more than twice as senstivity as STEMI criteria and much better than cardiologists. The patient was moved to the criticalcare area (stabilization room). This is the first version of the AI system. Accuracy was 91% and AUC was 0.95. Cath lab was activated.
Smith : there is some minimal ST elevation in V2-V6, but does not meet STEMI criteria. Transient STEMI has been studied and many of these patients will re-occlude in the middle of the night. Is it normal STE? The computer thinks so, and the physician thinks that is quite possible. However , there is terminal QRS distortion in lead V3.
Prehospital ECG: Obvious anterolateral STEMI (Proximal LAD occlusion) The cath lab was activated prehospital by the medics. Interventionalist at the Receiving Hospital: "No STEMI, no cath. Here is one case of a patient I saw. He was a 30-something with chest pain. A male in his 30's complained of sudden severe substernal chest pain.
We brought the patient into one of our criticalcare rooms and immediately got more history while recording this repeat ECG: The STE in I has greatly diminished or entirely disappeared. He wrote in his note that "The EKG showed early repolarization in I, V2-V3 but no clear STEMI pattern." We activated the cath lab.
I immediately activated the criticalcare team and walked the patient to the criticalcare area, our "Stabilization Room." This is why it is essential that the OMI/NOMI paradigm replace the STEMI/NonSTEMI paradigm. There are relatively large T-waves in V4-V6. Let's record another one." Learning Points: 1.
Submitted and written by Alex Bracey, with edits by Pendell Meyers and Steve Smith: I was walking through the criticalcare section of the ED when I overheard a discussion about the following ECG. 3) STEMI criteria failed to identify this acute coronary occlusion, like many others. What do you think?
There was high suspicion of OMI, so patient was brought to criticalcare area and another ECG was recorded just 7 minutes later as the pain had diminished to 4/10. Here is the repeat ECG at 52 minutes after arrival to triage: Obvious posterolateral STEMI Angiographic findings: 1. V5 and V6 have hyperacute T-waves.
He was brought to the criticalcare area where these rhythms were seen on the monitor: Wide complex tachycardia with no apparent P-waves, and very irregular Consistent with atrial fibrillation with aberrancy A Regular wide complex tachycardia. Instead, he complained of left chest "itchiness". LV Aneurysm? Would you give Thrombolytics?
This EKG was recorded as part of a standing order for criticalcare. He had been smoking an opiate and suddenly collapsed. He was ventilated with BVM on arrival. He awoke with naloxone. He denied any CP or SOB. An EKG was repeated at 5 minutes The T-wave is less hyperacute. Maybe there is some spontaneous reperfusion?
While calling for some help and arranging to have her transported to our criticalcare zone, I got this quick ultrasound which confirmed my suspicion: This quick view was all I was able to obtain in the circumstances. The cardiac monitor showed sinus rhythm but the automatic blood pressure cuff was not reading.
Here is his ED ECG: There is obvious infero-posterior STEMI. What are you worried about in addition to his STEMI? Comments: STEMI with hypokalemia, especially with a long QT, puts the patient at very high risk of Torsades or Ventricular fibrillation (see many references, with abstracts, below). Crit Care Med.
He reports that this chest pain feels different than prior chest pain when he had his STEMI/OMI, but is unable to further describe chest pain. Sensitivity was 87% for OMI in our validation study (it was 34% for STEMI criteria). He reports feeling nauseated with emesis. The Queen was not used in real time. Even the Queen can be wrong.
There is an obvious inferior STEMI, but what else? Besides the obvious inferior STEMI, there is across the precordial leads also, especially in V1. This STE is diagnostic of Right Ventricular STEMI (RV MI). In fact, the STE is widespread, mimicking an anterior STEMI. EKG is pictured below: What do you think?
Despite the clinical context, Cardiology was consulted due to concerns for a "STEMI". It is critically important for all EM and criticalcare providers to have an intimate understanding of hyperkalemia and its ECG findings. From Ken Grauer ( See below ) — with this Figure adapted from LITFL.
A middle aged patient who was 3 weeks s/p STEMI came from cardiac rehab where he developed some chest pain, dyspnea and weakness on the treadmill. There is no acute STEMI. This is diagnostic of recent, reperfused STEMI. This is diagnostic of recent, reperfused STEMI. Acute STEMI would have upright T-waves.
She had this ECG recorded: Obvious massive anterior STEMI She was quickly brought to the criticalcare area and the cath lab was activated. The blood pressure was 170/100 in the criticalcare area. Here is the ECG at 25 minutes: Terrible LAD STEMI (+) OMI So a CT scan was done which of course showed a normal aorta.
The PMCardio Queen of Hearts app asks you, before giving an interpretation of OMI ("STEMI-Equivalent"), whether the patient's clinical presentation is high risk for OMI. 20 cases with pseudonormalization Case continued The patient was moved to the criticalcare area, and cardiology was consulted.
It is diagnostic of OMI, but this is SUBACUTE OMI I sent this ECG to my "EKG Nerdz" friends, without any clinical info at all and they answered "OMI" The Queen said: "STEMI-Equivalent with High Confidence:" Notice she sees findings in both normal beats and PVCs. There is STE in V5-6. There are new Q-waves in aVL, V5-6.
Multidisciplinary criticalcare management of electrical storm. There was indication of parasympathetic overdrive ( the acute inferior STEMI with profound bradycardia and junctional escape ). [link] Jentzer, J. Noseworthy, P. Kashou, A. Chrispin, J., Tisdale, J. & Solomon, M. link] Mostofsky, E., Maclure, M., Sherwood, J.
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