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Suddencardiacdeath (SCD) remains a major health problem in all continents. It usually affects older patients suffering from coronaryarterydisease and cardiomyopathies. This group of patients defines the sudden arrhythmic death syndrome (SADS) where the cause of death is unknown.
Without immediate intervention, suddencardiacdeath can occur within minutes. This results in a failure to pump blood to the brain and other vital organs.
There's growing sentiment that drug-coated balloons could address an unmet clinical need among patients with coronaryarterydisease in the United States.” Each year, millions of people around the world undergo coronary angioplasty, a non-surgical intervention to treat blockages in the arteries that supply blood to the heart.
Atherosclerotic cardiovascular disease (ASCVD), caused by plaque buildup in arterial walls, is one of the leading causes of disability and death worldwide.1,2 alone, more than 800,000 of these people are at risk of MI and for approximately 200,000 of them, this may well be their second life-threatening cardiac event.
Prior research in suddencardiacdeath (SCD) genetics has focused on selected populations such as the young or “autopsy” negative. coronaryarterydisease [CAD]), which comprise the majority of SCD. These studies neglect older decedents or those with cardiovascular (CV) pathology (e.g.,
A large amount of endurance-exercise-related deaths occurred due to a cardiac incident during or following activity. Vigorous exercise is known to increase the risk of suddencardiacdeath. However, this increased risk manifests mostly among individuals with an underlying cardiacdisease.
It is crucial for every medical physician to recognize patients who are at risk of suddencardiacdeath due to their underlying medical condition. It is crucial for every medical physician to recognize patients who are at risk of suddencardiacdeath due to their underlying medical condition.
Cardiac catheterization revealed non-obstructive coronaryarterydisease. An external monitor revealed one episode of non-sustained supraventricular tachycardia, otherwise was unremarkable. He experienced worsening symptoms for which emergency department evaluation revealed anterior lead T-wave inversions on EKG.
In most cases, rather, the culprit is gross ischemia due to myocardial infarction, cardiomyopathy, or advanced coronaryarterydisease. Pleomorphic ventricular tachycardia and risk for suddencardiacdeath. genes implicated in congenital Long QT syndrome, such as SCN5A and KCNQ1). [1-3, 8] Liu, E.,
BackgroundRegardless of progress in treatment of coronaryarterydisease (CAD), there is still a significant residual risk of death in patients with CAD, highlighting the need for additional risk stratification markers. Journal of the American Heart Association, Ahead of Print. P=0.002) and 4.18 (95% CI, 1.5–11.4;P=0.005),
The authors are very careful to not use definitive causal terms, but it is pretty clear they strongly believe they are describing a new mechanism and novel etiology of suddencardiacdeath.
Recent research has shown that cardiovascular disease is higher in persons living with HIV compared with individuals without HIV, with an estimated 4-fold higher rate of suddencardiacdeath compared with the general population. million lives so far. Abd-Elmoniem, Ph.D., Abd-Elmoniem, Ph.D.,
Risk of acute myocardial infarction after the death of a significant person in ones life. Circumstances attending 100 suddendeaths from coronaryarterydisease with coroners necropsies. Circulation , 125 (3), 491496. link] Myers, A., & & Dewar, H. Heart , 37 (11), 11331143. link] Park, J.,
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