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Post cath ECG: Now there are hyperacute T-waves again, and recurrent ST depression in V2 This ECG would normally diagnostic of OMI until proven otherwise No further troponins were measured, but it looks like there is recurrent OMI Next day: A CT CoronaryAngiogram was done (CTCA) CARDIAC MORPHOLOGY AND FUNCTION: 1. IMPRESSION: 1.
Reversing or regressing coronary artery disease is possible. You cannot eliminate the plaque entirely, but multiple clinical trials have shown plaque regression using high-intensity cholesterol-lowering treatments, which I have discussed previously. REVERSAL Investigators. 2004 Mar 3;291(9):1071-80.
The most accurate way (But not the only way) to answer this question is whether or not you have plaque in your coronary arteries. If you already have plaque, your risk of event an event goes up proportional to the amount of plaque you have 2. So, low risk by anyone’s books.
We investigated the incidence of an acutely occluded coronary in patients presenting with STE-aVR with multi-lead ST depression. All electrocardiograms (ECGs) and coronaryangiograms were blindly analyzed by experienced cardiologists. BOTTOM Line from Today’s Case: As per Drs.
I suspect its use will rapidly accelerate given study after study now showing reductions in death, stent thrombosis, and nearly every other adverse outcome after PCI when intravascular imaging is used. Patients with severely calcified coronary lesions were randomized at 104 sites across the United States.
Smith Major Learning Point: The worst risk factor for a bad outcome in OMI is young age because cardiologists cannot believe that a young person can have an OMI. Only after her troponin peaked at 500,000 ng/L did she get her angiogram, which showed a 100% left main occlusion due to ruptured plaque. This gets drilled into them.
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