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Background:Epicardial patch defibrillators (EPDs) were commonly implanted in the 1990s for secondary prevention of sudden cardiac death. A repeat CT angiogram indicated contrast extension into the ventricular myocardium near the EPD but no lung spillage suggestive of pseudoaneurysm (Picture 1B).She
Finally, do a coronaryangiogram Possible alternative to pacing is to give a beta-1 agonist to increase heart rate. Because she has cardiomyopathy and ventricular dysrhythmias, the pacer included an Implanted Cardioverter-Defibrillator (ICD) Echo 6 days later after CRT: Normal estimated left ventricular ejection fraction.
Workup including routine laboratory results, 12-lead electrocardiogram (ECG), echocardiogram, and coronaryangiogram was non-specific. During the intravenous lacosamide infusion, the patient developed sudden cardiac arrest caused by ventricular arrhythmias necessitating resuscitation. 2893C>T, p.Arg965Cys) in the SCN5A gene.
The routine laboratory results, imaging study, coronaryangiogram, and echocardiogram (ECG) were normal. For secondary prevention, the patient underwent implantable cardioverter defibrillator implantation. The patient did not have underlying diseases. A type 1 BrS pattern was identified in one resting ECG.
After resuscitation and defibrillation , there were no more episodes of TdP. A coronaryangiogram was done that did not show significant coronary artery disease. Below is the patient’s 12 lead ECG following defibrillation. Post ROSC the patient was alert and cooperative. What does this ECG tell you?
Despite immediate chest compressions, and multiple rounds of defibrillation, he could not be resuscitated. Learning points 1] Acute Coronary Syndrome has many shades of clinical manifestation. He became unconscious as the monitor displayed VF.
During angiogram in the cath lab, the patient suffered two episodes of ventricular fibrillation for which he was successfully defibrillated. Angiogram showed a culprit lesion of 100% stenosis to the right coronary artery and 100% stenosis of the right posterior descending artery, both with TIMI 0 flow.
We can, therefore, put down the defibrillation pads, set aside the amiodarone, and look further at the ECG. The coronaryangiogram revealed no critical stenosis, or acute plaque ulceration. Paradoxically, though, the third green arrow identifies a QRS that is more narrow than the RBBB complexes surrounding it.
CT coronaryangiogram showed a hypoplastic RCA and dominant LCx. Most patients can be managed without and implantable cardioverter defibrillator (ICD) In patients with PVCs/VT and a presentation not typical for an idiopathic origin cardiac magnetic resonance (CMR) should be considered, even if the Echo is normal.
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