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Pulmonary sequestration (PS) is a rare congenital lung developmental anomaly characterized by abnormal lung tissue that receives its blood supply from the aorta. Subsequently, we placed vascular embolization devices to achieve closure. Among the various types, mixed PSrepresents a distinct subtype.
IntroductionVentricular septal defect (VSD) is a common congenital heart disease (CHD), accounting for 2030% of all CHD cases. No mortality or severe complications such as device embolization or complete atrioventricular block were observed. Data were analyzed using SPSS 20.0.ResultsThe
Inferior vena cava (IVC) agenesis is a rare congenital anomaly that has been implicated in up to 5% of unprovoked deep vein thrombosis (DVT) cases in young men under 30 years old. We present the case of a 28-year-old obese Caucasian male who arrived at our hospital with significant pain and swelling in his right lower extremity.
Patent Foramen Ovale (PFO) is a common congenital atrial septal defect present in 20%–35% of the general population. Although generally considered a benign anatomic variant, a PFO may facilitate passage of a thrombus from the venous to arterial circulation, thereby resulting in cryptogenic stroke or systemic embolization.
CT of the chest showed no pulmonary embolism but bibasilar infiltrates. It should be kept in mind that on occasions, beta-one agonist can result in increased ventricular ectopy e.g., in severe myocardial ischemia (by increasing myocardial demand), or sometimes with congenital long-QT syndrome. She was intubated.
Patent foramen ovale (PFO) is the most common congenital intracardiac defect that can cause right-to-left shunting and contribute to the paradoxical embolization of venous emboli, and even lead to stroke. Studies have shown that HES can affect the nervous system and may be associated with cerebral infarction.
Congenital Heart Defects Some individuals are born with heart defects that cause parts of the heart to work harder, leading to enlargement over time. Blood Clots: An enlarged heart is more prone to developing blood clots, which can lead to stroke or pulmonary embolism.
IntroductionVein of Galen Malformations (VOGM) are congenital brain arteriovenous malformations which often present during the neonatal or infantile periods. While endovascular embolization is the recommended management for this pathology, a subset of VOGM patients present with pathologic angiogenesis which complicates endovascular therapy.
1 While seen in both adult and pediatric populations, the majority of the VV‐AVFs seen in childhood are largely congenital, seen in the setting of connective tissue diseases such as Ehler‐Danlos Syndrome, Neurofibromatosis type 1, and Marfan’s syndrome, with abnormal involution of the proatlantal system also thought to be implicated.2,3,4
Introduction:Vein of Galen malformations (VOGM) are challenging congenital arteriovenous malformations (AVMs) to treat. Stroke, Volume 56, Issue Suppl_1 , Page A132-A132, February 1, 2025. During management, for unknown reasons, VOGM may develop fine angiogenic networks of feeding vessels within subarachnoid spaces.
Introduction:Vein of Galen Malformations (VOGM) are congenital arteriovenous malformations which carry a mortality of 76.7% Outcomes have improved over time with advances in endovascular embolization. Stroke, Volume 55, Issue Suppl_1 , Page AWP221-AWP221, February 1, 2024. in untreated patients.
stroke), peripheral arterial disease, congenital heart anomalies, deep vein thrombosis, and pulmonary embolism. Cardiovascular diseases (CVDs) encompass a range of disorders affecting the heart and blood vessels, such as coronary heart disease, cerebrovascular disease (e.g.,
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