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Heart disease remains one of the leading causes of death worldwide, often attributed to a mix of lifestyle choices, environmental factors, and geneticpredispositions. While you can’t change your genetic code, having a family history of heart disease does not mean your fate is sealed.
Circulation, Ahead of Print. Over the past decade, new research has advanced scientific knowledge of neurodevelopmental trajectories, factors that increase neurodevelopmental risk, and neuroprotective strategies for individuals with congenital heart disease.
Circulation, Volume 150, Issue Suppl_1 , Page A4112869-A4112869, November 12, 2024. This innovative method allowed for the efficient synthesis and visualisation of complex data.Results:The SRPS table methodically organizes riskfactors into low (0-2 points), moderate (3-5 points), and high (6+ points) categories.
Circulation: Arrhythmia and Electrophysiology, Ahead of Print. The highest HRs (95% CIs) of AF were observed for participants at high geneticrisk who consumed >2 L/wk of ASB (HR, 3.51 [95% CI, 2.94–4.19]), PJ consumption ≤1 L/wk was associated with a modestly lower risk for AF.
Circulation: Genomic and Precision Medicine, Volume 17, Issue 6 , Page e004685, December 1, 2024. The female-specific positive association of PGSMDwith CAD risk was replicated in BioVU.CONCLUSIONS:Genetic predisposition to MD confers a greater risk of CVDs in females versus males, even in the absence of any depression diagnosis.
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