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A teenager involved in a motor vehicle collision with abnormal ECG

Dr. Smith's ECG Blog

See these publications for more information Overall, management for cardiac contusion is mostly supportive unless surgical complications develop, involving appropriate treatment of dysrhythmias and hemodynamic instability. Circulation: Cardiovascular Imaging. 2015, March 1). Myocardial contusion in an 8-year-old boy. Cramer, M.

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A man in his 70s with weakness and syncope

Dr. Smith's ECG Blog

There were no dysrhythmias on cardiac monitor during observation. This discussion comes from this previous post: Hyperthermia and ST Elevation Discussion Brugada Type 1 ECG changes are associated with sudden cardiac death (SCD) and the occurrence of ventricular dysrhythmias. Circulation, 117, 1890–1893. [3]:

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A Relatively Narrow Complex Tachycardia at a Rate of 180.

Dr. Smith's ECG Blog

I also believe that we physicians and medics are eager to treat dysrhythmias, and we want to see them even when they are not there. Circulation 2008;117(14):1897-907. Dilated pupils and hypertension are a strong clue to sympathetic overload, but don't forget anticholinergic syndromes, including tricyclics! Pollan S, Tadjziechy M.

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How to wean off Beta-blockers

Dr. Sanjay Gupta

Similarly, there have been case reports of patients who were taking beta blockers to control dangerous heart rhythm disturbances and when the beta blocker was abruptly stopped it led to precipitation of ventricular dysrhythmias and even in some cases death. This is termed as ‘Acute beta blocker withdrawal syndrome’.

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Patient with severe DKA, look at the ECG

Dr. Smith's ECG Blog

So the real QT is shorter, but the computer does not mention the U-wave, and the U-wave is as important as the T-wave in predicting cardiac dysrhythmias. mEq of K pushed fast and circulated theoretically would raise serum K immediately by 1.0 This is an extremely dangerous ECG. The K returned at 1.9 This is extremely low for DKA.

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STEMI with Life-Threatening Hypokalemia and Incessant Torsades de Pointes

Dr. Smith's ECG Blog

Could the dysrhythmias have been prevented? mEq of K pushed fast and circulated theoretically would raise serum K immediately by 1.0 Severe hypokalemia in the setting of STEMI or dysrhythmias is life-threatening and needs very rapid treatment. Since it takes some time (how long?) mEq/L, and 10 mEq would increase it by 3.3

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An athletic 30-something woman with acute substernal chest pressure

Dr. Smith's ECG Blog

Now you have ECG and troponin evidence of ischemia, AND ventricular dysrhythmia, which means this is NOT a stable ACS. Circulation. Circulation, 137(19), p.e523. (For those of you who are accustomed to the units of high sensitivity troponin (ng/L), this is equivalent to 20,956.00 Again, cath lab was not activated. 112.105718.

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