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Coronary artery spasm (CAS), or Prinzmetal angina, is a recognised cause of myocardial ischaemia in non-obstructed coronary arteries which typically presents with anginal chestpain. The patient presented with recurrent palpitations and pre-syncope, with no chestpain.
Written by Pendell Meyers Two patients with acute chestpain. Patient 1: Patient 2: Patient 1: A man in his 40s with minimal medical history presented with acute chestpain radiating to his R shoulder. Two patients with chestpain. Do either, neither, or both have OMI and need reperfusion?
A 50-something male had onset of chestpain 1 hour prior to ED arrival. Endorses some associated SOB, but denies back pain, fever, cough, chills, leg swelling, or other new symptoms. Always get serial ECGs in a patient with acute chestpain. It is constant, 9/10, left-sided CP that radiates into left arm and jaw.
The increased use of radiofrequency ablation (RFA) for atrial fibrillation (AF) has led to a rise in cases of pulmonary vein stenosis or occlusion (PVS/O) as a complication. Clinical signs of PVS/O post-ablation can vary from no symptoms to common respiratory issues like coughing, hemoptysis, shortness of breath, and chestpain.
A 50-something male with hypertension and 20- to 40-year smoking history presented with 1 week of stuttering chestpain that is worse with exertion, which takes many minutes to resolve after resting and never occurs at rest. At times the pain does go to his left neck. It is a ssociated with mild dyspnea on exertion.
The patient presented to an outside hospital An 80yo female per triage “patient presents with chestpain, also hurts to breathe” PMH: CAD, s/p stent placement, CHF, atrial fibrillation, pacemaker (placed 1 month earlier), LBBB. HPI: Abrupt onset of substernal chestpain associated with nausea/vomiting 30 min PTA.
Despite this, the patient went on to develop chestpain, which was accompanied by electrocardiographic signs of acute extensive anterior wall myocardial infarction and elevated troponin I levels.
Until now, patients with aortic stenosis—a narrowing of one of the heart's main valves—have had to wait until symptoms become severe before undergoing valve replacement.
Written by Jesse McLaren, comments by Smith A 55 year old with a history of NSTEMI presented with two hours of exertional chestpain, with normal vitals. See these posts: ChestPain, ST Elevation, and an Elevated Troponin: Should we Activate the Cath Lab? What do you think?
A 60-something yo female presented w/ exertional chestpain for 3 days. Pain was 8/10 and constant. She has been experiencing progressively worsening exertional dyspnea and chest tightness mostly when climbing up flights of stairs since early September. But the patient has active chestpain.
A 40-something male presented by ambulance with one hour of chestpain that was improving after sublingual nitroglycerine and 325 mg of aspirin, chewed. Here is the angiogram: --Culprit is 100% stenosis in the proximal RCA. (It Here is his initial ED ECG: What do you think? Then the ED doc would be dependent on that first ECG.
A 40-something with severe diabetes on dialysis and with known coronary disease presented with acute crushing chestpain. 2 years prior he had an angiogram which showed 90% proximal stenosis of the circumflex. The pain did not resolve with NTG, and so he went to emergent angiography: 1. Left main: no obvious stenosis.
Shortly after receiving epinephrine, the patient developed new leg cramps and chestpain. The chestpain was described as sharp and radiated to both arms. During active chestpain an ECG was recorded: Meyers ECG interpretation: Sinus tachycardia, normal QRS complex, STD in V2-V6, I, II, III and aVF.
Sent by Drew Williams, written by Pendell Meyers A man in his 50s with history of hypertension was standing at the bus stop when he developed sudden onset severe pressure-like chestpain radiating to his neck and right arm, associated with dyspnea, diaphoresis, and presyncope. EMS arrived and administered aspirin and nitroglycerin.
Submitted by anonymous, written by Pendell Meyers A woman in her 50s presented to the Emergency Department with chestpain and shortness of breath that woke her from sleep, with diaphoresis. See these other cases of arterial pulse tapping artifact: A 60 year old with chestpain Are these Hyperacute T-waves?
The ACC/AHA guidelines mandate less than 2 hours cath for patients with ACS with refractory pain, pulmonary edema, or electrical or hemodynamic instability. Angiogram at 4 hours after ECG 1 (and approximately 6 hours after pain onset): Culprit is 100% stenosis in the mid RCA. No wall motion abnormality.
Sent by anonymous, written by Pendell Meyers A man in his 50s with no prior known medical history presented to the Emergency Department with severe intermittent chestpain. He denied any lightheadedness, shortness of breath, vomiting, or abdominal pain. Isn't it amazing?? Don’t Ignore Bedside Echo Results! —
Written by Pendell Meyers A middle aged man called EMS for acute chestpain. He had 50% stenosis of the LAD which was deemed not culprit, and all other vessels were normal. EMS recorded this ECG during active symptoms and transmitted it to the ED: I had no information when I was shown the ECG. I said "Not OMI. No intervention.
But syncope or seizure alone, without chestpain, is not enough to call it Wellens syndrome. Without chestpain, the pretest probability is not very high. Further review of systems with the patient fully awake revealed that the patient had been having chestpain on and off all week.
Submitted and written by Megan Lieb, DO with edits by Bracey, Smith, Meyers, and Grauer A 50-ish year old man with ICD presented to the emergency department with substernal chestpain for 3 hours prior to arrival. At this time he reported ongoing chestpain and was given aspirin and nitroglycerin.
Upon questioning patient, he denies having any chestpain or chest tightness of any sort. In the absence of chestpain and negative troponin , it appears less likely that he is having acute coronary syndrome though EKG appears concerning. Pericarditis would be even more unlikely in someone without chestpain.
A 63 year old man with a history of hypertension, hyperlipidemia, prediabetes, and a family history of CAD developed chestpain, shortness of breath, and diaphoresis after consuming a large meal at noon. He called EMS, who arrived on scene about two hours after the onset of pain to find him hypertensive at 220 systolic.
A middle aged male presented at midnight after 14 hours of constant, severe substernal chestpain, radiating to his throat and to bilateral jaws, and associated with diaphoresis. The pain was not positional, pleuritic, or reproducible. It was not relieved by anything. He had no previous medical history. Is it STEMI or NonSTEMI?
Written by Jesse McLaren, with edits from Smith A 30 year old with a history of diabetes presented with two days of intermittent chestpain and diaphoresis, which recurred two hours prior to presentation. The chestpain was refractory to nitro so the cath lab was activated: 100% proximal LAD and 99% mid circumflex occlusions.
Sent by Anonymous, written by Pendell Meyers A man in his 60s with history of CAD and 2 prior stents presented to the ED complaining of acute heavy substernal chestpain that began while eating breakfast about an hour ago, and had been persistent since then, despite EMS administering aspirin and nitroglycerin. Pre-intervention.
There is a patient with persistent chestpain and an initial troponin I over 52 ng/L; 52 ng/L has an approximate 70% PPV for acute type I MI in a chestpain patient. Pain was severe and persistent. CT angiography chest assessing for PE and dissection negative. Heparin drip was initiated. Normal RV function.
He had suffered a couple bouts of typical chestpain in the last 24 hours. This ECG (ECG #3) was recorded immediately after the last episode of pain spontaneously resolved. The pain had lasted about one hour. Case A 40-something male presented to triage. There are classic Wellens' waves in V2-V5. Am Heart J.
Submitted by Ali Khan MD and James Mantas MD, MS, written by Pendell Meyers A man in his 50s with history of diabetes, hypertension, and tobacco use presented to the ED with 24 hours of worsening left sided chestpain radiating to the back, characterized as squeezing and pinching, associated with shortness of breath.
In a 57-year-old man with chestpain, an ECG obtained by EMS showed widespread ST-segment depressions. At the hospital, left main coronary-artery stenosis was seen on angiography (shown in a video).
Since the pathologist does not know the original cross-sectional area of the artery or the amount of compensatory enlargement of the artery from evaluation of a single cross section of the artery at a site of stenosis, the degree of luminal narrowing of that segment cannot be determined. These are typical findings at sites of plaque rupture.
Collectively, this study shows that Cleerly AI-QCT ISCHEMIA , when used in conjunction with Cleerly LABS, can provide a 3-in-1 approach for the assessment of atherosclerosis, stenosis and ischemia. JACC Cardiovasc Imaging. 2024 Feb 29:S1936-878X(24)00039-1. doi: 10.1016/j.jcmg.2024.01.007. 2024.01.007. Epub ahead of print. PMID: 38483420.
Sent by anonymous A man in his 40s with no previous heart disease presented within 30 minutes of onset of acute chestpain that started while exercising. Angiogram findings included: 95% mid RCA stenosis with occluded distal right PDA secondary to thrombus (peristent OMI). Chestpain and a computer ‘normal’ ECG.
There was no chestpain. V1 and V2 are probably placed too high on the chest given close morphological similarity to aVR. The LM has an irregular 30% distal stenosis, followed by an 80% ostial LAD stenosis, and total occlusion of the LAD proximally with TIMI grade 1 flow in the distal vessel. Type II ischemia.
A 20-something male presented from an outside facility with Chestpain. He came with this ECG from the outside facility, recorded 1 hour after pain onset: There is at least 2 mm of inferior ST elevation, with reciprocal ST depression in aVL, ST flattening in V4-V6, and T-wave inversion in V2. Vital signs were normal.
A 60-something awoke with 10/10 crushing chestpain. The angiogram showed an open artery with 95% stenosis and thrombosis and it was stented. But the patient's chestpain continues and so you order a 2nd ECG (ECG 2 here). He walked in to triage. I would expect that a stent would be placed.
Methods This was a retrospective, observational, single-center study of 403 patients with typical or atypical angina-like chestpain undergoing acetylcholine (ACH) spasm provocation testing and OCAD. We defined positive epicardial spasm as ≥90% transient stenosis and usual chest symptoms or ischemic ECG changes.
Here I annotate it: This shows 100% occluded circumflex (red arrow) and a 90% stenosis of the LAD (Yellow arrow). The LAD was thought to be not thrombotic, but a chronic tight stenosis. My THOUGHTS on ECG #1: We are told that the patient in today’s case had an episode of severe chestpain 3 nights prior to admission.
He reports significant chestpain at the base of his scapula on the right side along with new shortness of breath. Wellen's waves indicate that, when the patient was having chestpain, there was occlusion. See these casese (and I have many others): First ED ECG is Wellens' (pain free). A 70-something y.o.
BUT — Cardiac catheterization done a little later did not reveal any significant stenosis. Despite the absence of significant coronary stenosis on her post-arrest cath — the ECG in Figure-1 is clearly diagnostic of an extensive anterolateral STEMI ( presumably from acute LAD [ L eft A nterior D escending ] coronary artery occlusion).
On the second morning of his admission, he developed 10/10 chestpain and some diaphoresis after breakfast. The patient was given opiates which improved his chestpain to 7/10. The consulting cardiologist wrote in their note: “Could be cardiac chestpain. She is usually incredibly good at recognizing them!
Written by Pendell Meyers A man in his early sixties with no significant medical history (including a "negative cardiac workup a few years ago" for unclear indication) called 911 for acute chestpain constantly for the past 5 hours. However, this patient has active chestpain, and thus this this is inferior-posterior OMI.
He arrived to the ED by helicopter at 1507, about three hours after the start of his chestpain while chopping wood around noon. He arrived to the ED by ambulance at 1529, only a half hour after the start of his chestpain around 1500 while eating. Angiography revealed a 30% nonobstructive stenosis of the mid LAD.
Here is the clinical story: A 40 year old male with no cardiac history presented with acute substernal chestpain that started 40 minutes prior to arrival. In spite of a relatively short QTc of 376 ms, the very low R-wave amplitude in V4 and the ST Elevation at 60 ms after the J-point in lead V3 contribute to a high final value.
He presented with chestpain, not relieved by nitro, pain reproducible on exam and centered around the pacemaker insertion site. Here is his ECG one month prior, on admission for chestpain at that time also: Similar ratios. Here is his ED ECG There is RV Pacing. How could you suspect that this is false positive?
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