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Coronary artery spasm (CAS), or Prinzmetal angina, is a recognised cause of myocardial ischaemia in non-obstructed coronary arteries which typically presents with anginal chestpain. This case report describes an atypical presentation of CAS in a 68-year-old white British male with cardiovascular riskfactors.
By Magnus Nossen This ECG is from a young man with no riskfactors for CAD, he presented with chestpain. The patient is a young adult male with chestpain. The chestpain was described as pressure like and radiation to both arms and the jaw. How would you assess this ECG?
Written by Willy Frick A man in his early 40s with BMI 36, hypertension, and a 30 pack-year smoking history presented with three days of chestpain. He described it as a mild intensity, nagging pain on the right side of his chest with nausea and dyspnea. It started while he was at rest after finishing a workout.
What are the most useful historical factors to increase and decrease your pretest probability for ACS? Which cardiac riskfactors have predictive value for ACS? The post Ep 128 Low RiskChestPain and High Sensitivity Troponin – A Paradigm Shift appeared first on Emergency Medicine Cases.
A healthy 45-year-old female presented with chestpain, with normal vitals. The patient was previously healthy, with no atherosclerotic riskfactors, and developed chestpain after an episode of stress. The pain was crushing retrosternal, radiated to the arms and was associated with lightheadedness.
Healthy male under 25 years old with a pretty good story for acute onset crushing chestpain relieved with nitro. Aggressive riskfactor modification. PEARL: Most patients who present with new chestpain + ECG changes + positive troponin — will not need Cardiac MRI. No pericardial effusion on ultrasound."
Background:Patients with de novo chestpain, referred for evaluation of possible coronary artery disease (CAD), frequently have an absence of CAD resulting in millions of tests not having any clinical impact. Circulation: Genomic and Precision Medicine, Volume 16, Issue 5 , Page 442-451, October 1, 2023.
A 63 year old man with a history of hypertension, hyperlipidemia, prediabetes, and a family history of CAD developed chestpain, shortness of breath, and diaphoresis after consuming a large meal at noon. He called EMS, who arrived on scene about two hours after the onset of pain to find him hypertensive at 220 systolic.
This condition reduces blood flow to the heart, increasing the risk of angina (chestpain) and heart attacks. Coronary Artery Disease (CAD) : High blood pressure accelerates the development of CAD by promoting the buildup of plaques in the coronary arteries.
The National Institute for Health and Care Excellence (NICE) advise against routine testing for coronary artery disease (CAD) in patients with non-anginal chestpain (NACP). Cardiovascular riskfactors were compared between the groups. Over 23 months, 866 patients with NACP underwent CTCA. We found 11.5%
Submitted by Ali Khan MD and James Mantas MD, MS, written by Pendell Meyers A man in his 50s with history of diabetes, hypertension, and tobacco use presented to the ED with 24 hours of worsening left sided chestpain radiating to the back, characterized as squeezing and pinching, associated with shortness of breath.
Objective Prehospital rule-out of non-ST-segment elevation acute coronary syndrome (NSTE-ACS) in low-risk patient with a point-of-care troponin measurement reduces healthcare costs with similar safety to standard transfer to the hospital. Low-risk patients (HEAR score ≤3) were included.
A 20-something male presented from an outside facility with Chestpain. No thromboembolism risks, not pleuritic, no radiation to the back. No cardiac riskfactors, no cocaine use. Vital signs were normal. History: Onset of CP 2.5 hours prior to ED arrival. Tight and pressure, radiates to right arm, + nausea, + SOB.
IntroductionPatients with cognitive impairment often have a history of cardiovascular disease (CVD) or multiple cardiovascular riskfactors (CRFs) such as hypertension, obesity, and hypercholesterolemia. The literature reports that CVD with CRFs may increase the risk of developing vascular dementia and Alzheimer’s Disease.
On the second morning of his admission, he developed 10/10 chestpain and some diaphoresis after breakfast. The patient was given opiates which improved his chestpain to 7/10. The consulting cardiologist wrote in their note: “Could be cardiac chestpain. She is usually incredibly good at recognizing them!
Because its symptoms are commonly associated with so many other conditions, like a cough and chestpain, lung cancer can be hard to diagnose. At CTVS, we are proponents of early screening for lung cancer, especially if you have certain riskfactors such as… Source
Genetic protein S (PS) deficiency caused by PROS1 gene mutation is an important riskfactor for hereditary thrombophilia.Case introductionIn this case, we report a 28-year-old male patient who developed a severe pulmonary embolism during his visit. The patient had experienced one month of chestpains, coughing and hemoptysis symptoms.
Submitted and written by Alex Bracey with edits by Pendell Meyers and Steve Smith Case A 50ish year old man with a history of CAD w/ prior LAD MI s/p LAD stenting presented to the ED with chestpain similar to his prior MI, but worse. The pain initially started the day prior to presentation. The ST elevation from today is ~0.2
years]) admitted to the China ChestPain Center Database between 2016 and 2021. Hierarchical clustering of 15 medical conditions was performed to derive multimorbidity patterns. The primary outcome was a composite of inhospital adverse events.
Likelihood of CAD can be estimated based on symptoms and riskfactors, and an abnormal ECG may also be helpful. Patients with moderate/high probability of CAD are usually referred for further tests including angiography (CT or invasive) or isotope-based myocardial perfusion scans.
Understanding the riskfactors, recognizing the signs and seeking guidance from a cardiologist can play a significant role in preventing and treating this disease. Identifying Those at Risk for Heart Disease Heart disease describes a range of disorders that affect the cardiovascular system and the heart.
Written by Pendell Meyers A man in his late 40s with several ACS riskfactors presented with a chief complaint of chestpain. Several hours prior to presentation, while driving his truck, he started experiencing new central chestpain, without radiation, aggravating/alleviating factors, or other associated symptoms.
Unhealthy Coping Mechanisms When under constant stress, many people turn to unhealthy coping mechanisms like overeating, smoking, or excessive alcohol consumption – habits that can further exacerbate cardiovascular issues by contributing to obesity, high blood pressure, and other riskfactors.
She did not report any chestpain or pressure. She was brought to the Emergency Department and this ECG was recorded while she was still feeling nauseous but denied chestpain, shortness of breath, or other symptoms: What do you think? A male in his 60s with chestpain A Male in his 60s with Chestpain.
Minimize your intake of processed foods, sugary snacks, and foods high in saturated and trans fats, as these can contribute to heart disease riskfactors such as high cholesterol and hypertension. By reducing stress levels, you can lower your risk of heart disease and improve overall cardiac health.
A 39 yo otherwise healthy man with no riskfactors was walking at the mall when he developed chest pressure. However, if you notice the ST depression, you then realize that this is ischemic chestpain, not esophageal spasm. He presented to the ED after 30 minutes, now also feeling weak. He was diaphoretic.
Riskfactors such as smoking, chronic kidney disease, and aging can contribute to plaque formation. The gradual loss of elasticity in arterial walls and the presence of other riskfactors, such as high blood pressure and diabetes, contribute to the increased risk of cardiovascular disease (CVD) observed in aging populations.
female with HTN, HLD, diabetes, ESRD on dialysis is brought in by EMS with sudden onset, left -sided chestpain for the past four hours. While she was in her bed at home, she had sudden onset of left sided chestpain that radiated to her shoulder. The pain was pleuritic, without nausea or diaphoresis.
Important riskfactors for diseases of blood vessels are high blood pressure and sugar, high blood fat levels, overweight/obesity, and smoking. Advancing age, heredity and male gender are also riskfactors for diseases of blood vessels. Blood seeps into the wall of the aorta and spreads along the wall of the aorta.
As discussed in detail in ECG Blog #228 — this seemingly qualifies as a “ Silent ” MI ( Approximately half of those MIs not accompanied by CP — have some other associated symptom such as syncope, which substitutes as a “chestpain equivalent” ). Longterm prognosis of patients with MINOCA clearly depends on the underlying etiology.
This case was recently posted by Tyron Maartens on Facebook EKG club (he agreed to let me post it here), with the following clinical information: "42 year old male with two weeks of intermittent chest discomfort, awoke 4 hours prior to this ECG with a more severe, heavy chestpain (5/10). BP 112/80, SpO2 100%.
Written by Pendell Meyers A male in his early 50s presented with waxing and waning chestpain starting at rest. He had multiple cardiovascular riskfactors and the EM physician strongly suspected ACS. Here is his initial ECG: What do you think? mm of the "required" 1.0
A middle aged male with no h/o CAD presented with one week of crescendo exertional angina, and had chestpain at the time of the first ECG: Here is the patient's previous ECG: Here is the patient's presenting ED ECG: There is isolated ST depression in precordial leads, deeper in V2 - V4 than in V5 or V6. There is no ST elevation.
Meyers and Smith, who instantly recognized impending LAD occlusion Although I completely agreed on the need for prompt cardiac cath in this 50s man with riskfactors and new symptoms — I did not initially recognize the “culprit artery”. Sometimes serial ECGs minimizes the delay. Sometimes serial ECGs minimizes the delay. CREDIT to Drs.
She asked me why I felt she had had a heart attack and I explained to her that she had had chestpains and the blood test indicating damage to the heart was elevated and that was all we needed to say that she had had a heart attack. Genetics and physiological stress are also riskfactors.
AFib causes a variety of symptoms, including fast or chaotic heartbeat, fatigue, shortness of breath, and chestpain, and causes about 450,000 hospitalizations each year, according to the Centers for Disease Control and Prevention. For example, kidney disease is not included in CHA 2 DS 2 -VASc.
A male in his 60's called 911 for chestpain. He had some cardiac riskfactors including hypertension, on meds, but no previous coronary disease. His pain was intermittent and he was vague about when it was present and when it was resolved. Here is his prehospital ECG: Diagnosis?
As age advances and depending on riskfactors like heredity, smoking, high blood pressure and diabetes, fat build up occurs in these blood vessels. This can block smooth flow of blood and the person can develop chestpain. Coronary arteries are blood vessels supplying oxygenated blood to the heart.
"I have enclosed the ECG from a 50-something year old male who complained of chestpain. He had no further riskfactors for atherosclerosis besides hypertension. For clarity — I’ve labeled the compressed 12-lead tracing of this 50-ish year old man with new chestpain ( Figure-1 ).
A 40 something woman with a history of hyperlipidemia and additional riskfactors including a smoking history presented with substernal chestpain radiating to "both axilla" as well as the upper back. She was reportedly "pacing in her room while holding her chest". The source of this case is anonymous.
hours of substernal chest pressure. She had zero CAD riskfactors. SCAD occurs in patients with few or non-traditional cardiovascular riskfactors. The patient profile in this case is deceptive = a seemingly healthy, athletic and very fit 30-year woman with no riskfactors and no significant prior medical history.
A 70-something female with no previous cardiac history presented with acute chestpain. She awoke from sleep last night around 4:45 AM (3 hours prior to arrival) with pain that originated in her mid back. She stated the pain was achy/crampy. Over the course of the next hour, this pain turned into a pressure in her chest.
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The patient contacted EMS after a few hours of chestpain that started 5:30 AM. The pain was described as 6/10 radiating to the right shoulder. The chestpain was described as both sharp and pressure like. There is slight ST depression in lateral chest leads V4,V5,V6 — but I thought this to be nonspecific.
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