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This is a previously healthy male teenager who was awoken by chestpain. The pain is described as located in the midsternal area, radiating to the right arm, described as 8-9/10 and worse with deep inspirations. In the evening, he became diaphoretic and complained of 9/10 continuous chestpain.
He denied chestpain or shortness of breath. In the clinical context of weakness and fever, without chestpain or shortness of breath, the likelihood of Brugada pattern is obviously much higher. Pediatric and elderly patients were more predisposed to developing an arrhythmic event in the setting of fever [7].
There was no chestpain. Extensive conduction system abnormalities can have various causes (ischemia, genetic, infectious, amyloid, etc). This was written by Magnus Nossen The patient is a female in her 50s. She presented with a one week hx of «dizziness» and weakness. She was feeling fine prior to the last seven days.
It was from a patient with chestpain: Note the obvious Brugada pattern. Pediatric and elderly patients were more predisposed to developing an arrhythmic event in the setting of fever [7]. The elevated troponin was attributed to either type 2 MI or to non-MI acute myocardial injury. There is no further workup at this time.
She did not even need to ask in this case, because even if the patient presented with chestpain, she would call it NEGATIVE. This ST-T wave pattern in lead V5 is not seen in other leads, as would be expected if this was truly a change of acute ischemia. What about the R = S Phenomenon in the Inferior Leads?
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