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There was apparently no syncope and he had no bony injuries, but he did complain of left sided chestpain. His chest was tender. Is there STEMI? A bedside cardiac ultrasound was normal. An ECG was recorded: Avinash was understandably confused by this ECG. He wrote: "ECG 1 - shows wide ???IVCD IVCD type rhythm ??
A 90 yo with a history of orthostatic hypotension had a near syncopal event followed by chestpain. Chestpain was resolved upon arrival in the ED. In other words, after reperfusion therapy for STEMI, the appearance of AIVR is usually a good sign, meaning that the artery is reperfused. His previous ECG was normal.
He denied chestpain or shortness of breath. In the clinical context of weakness and fever, without chestpain or shortness of breath, the likelihood of Brugada pattern is obviously much higher. There were no dysrhythmias on cardiac monitor during observation. See below for PM Cardio digitized version of this.
A late middle-aged man presented with one hour of chestpain. Here is his ED ECG: There is obvious infero-posterior STEMI. What are you worried about in addition to his STEMI? Could the dysrhythmias have been prevented? to greatly decrease risk (although in STEMI, the optimal level is about 4.0-4.5
Written by Pendell Meyers and Peter Brooks MD A man in his 30s with no known past medical history was reported to suddenly experience chestpain and shortness of breath at home in front of his family. Chestpain, SOB, Precordial T-wave inversions, and positive troponin. What is the Diagnosis? Now another, with ultrasound.
It is equivalent to a transient STEMI. Now you have ECG and troponin evidence of ischemia, AND ventricular dysrhythmia, which means this is NOT a stable ACS. As emphasized by Dr. Smith — the best way to demonstrate acute ischemia is by identifying d ynamic S T- T wave c hanges in association with change in the nature of chestpain.
Opinions vary widely on the K level at which a patient must be admitted on a monitor because of the risk of ventricular dysrhythmias. My rationale is that if the K is affecting the ECG, then it is affecting the electrical milieu and can result in serious dysrhythmias. Until some real data is available, my opinion is this: 1.
The medics were worried about STEMI, as it meets STEMI criteria. He was admitted for monitoring, as his risk of a ventricular dysrhythmia as cause of the syncope is high ( very high due to HFrEF and ischemic cardiomyopathy ). He denied chestpain or dyspnea throughout. What do you think? There is LVH.
2) The STE in V1 and V2 has an R'-wave and downsloping ST segments, very atypical for STEMI. Cardiology was consulted and they agreed that the EKG had an atypical morphology for STEMI and did not activate the cath lab. It was from a patient with chestpain: Note the obvious Brugada pattern. Bicarb 20, Lactate 4.2,
It was edited by Smith CASE : A 52-year-old male with a past medical history of hypertension and COPD summoned EMS with complaints of chestpain, weakness and nausea. Clinical Course The paramedic activated a “Code STEMI” alert and transported the patient nearly 50 miles to the closest tertiary medical center. What do you see?
A 26 year old male presented with syncope and chestpain. Smith : I recognize this as a STEMI mimic. No signs of OMI" The chestpain resolved after some time, and another ECG was recorded: The ST Elevation is nearly gone. He was admitted for monitoring and had no dysrhythmias. I was not alarmed.
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