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This is an interesting case for your students who want to delve into dysrhythmias with an eye on detail. I will start the discussion by admitting that I am not an expert of electrophysiology or complex dysrhythmias. I hope some of our dysrhythmia Gurus will delve into the rhythm and maybe even provide laddergrams.
This is an interesting case for your students who want to delve into dysrhythmias with an eye on detail. I will start the discussion by admitting that I am not an expert of electrophysiology or complex dysrhythmias. I hope some of our dysrhythmia Gurus will delve into the rhythm and maybe even provide laddergrams.
Written by Bobby Nicholson MD and Pendell Meyers A man in his 30s presented to the ED for evaluation of chestpain and palpitations. At this point, the patient had been symptomatic for almost 5 hours, appeared unwell with chestpain and diaphoresis. Thus, the patients rhythm is atrial fibrillation with WPW.
He denied chestpain or shortness of breath. In the clinical context of weakness and fever, without chestpain or shortness of breath, the likelihood of Brugada pattern is obviously much higher. There were no dysrhythmias on cardiac monitor during observation. See below for PM Cardio digitized version of this.
A late middle-aged man presented with one hour of chestpain. Could the dysrhythmias have been prevented? Severe hypokalemia in the setting of STEMI or dysrhythmias is life-threatening and needs very rapid treatment. Most recent echo showed EF of 60%. He also had a history of chronic kidney disease, stage III.
It was from a patient with chestpain: Note the obvious Brugada pattern. She has not yet been seen by electrophysiology or had further genetic testing for Brugada syndrome. The elevated troponin was attributed to either type 2 MI or to non-MI acute myocardial injury. There is no further workup at this time.
Check : [vitals, SOB, ChestPain, Ultrasound] If the patient has Abdominal Pain, ChestPain, Dyspnea or Hypoxemia, Headache, Hypotension , then these should be considered the primary chief complaint (not syncope). Electrophysiologic studies were performed in selected patients only as clinically appropriate.
This middle-aged man with no cardiac history but with significant history of methamphetamin and alcohol use presented with chestpain and SOB, worsening over days, with orthopnea. Patient course The patient was started on beta blockers and schedule for an electrophysiologic study. BP:143/99, Pulse 109, Temp 37.2 °C
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