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This was sent by anonymous The patient is a 55-year-old male who presented to the emergency department after approximately 3 to 4 days of intermittent central boring chestpain initially responsive to nitroglycerin, but is now more constant and not responsive to nitroglycerin. It is unknown when this pain recurred and became constant.
Sent by Magnus Nossen MD, written by Pendell Meyers A man in his 50s, previously healthy, developed acute chestpain. The primary care physician there evaluated this patient and deemed the chestpain to be due to gastrointestinal causes. The ECG was also interpreted as normal by the primary care physician.
Click here to sign up for Queen of Hearts Access Case A 58-year-old woman presented to the ED with burning chestpain that started 2-3 hours earlier while sitting on a porch swing. In any case, it is diagnostic of OMI in a chestpain patient. She was successfully defibrillated and taken back to the ED.
A patient had a cardiac arrest with ventricular fibrillation and was successfully defibrillated. IF the initial ECG following successful defibrillation shows evidence of acute OMI — such patients have much to gain from immediate cath with PCI. As per Dr. Smith — the intuitive answer should be obvious.
Primary preventive therapies, such as implantable cardioverter defibrillators, are not personalized and not predictive. Methods We retrospectively analyzed the measurements at rest for 191 patients with acute chestpain (ACP) magnetocardiographically.
A healthy 45-year-old female presented with chestpain, with normal vitals. The patient was previously healthy, with no atherosclerotic risk factors, and developed chestpain after an episode of stress. The pain was crushing retrosternal, radiated to the arms and was associated with lightheadedness.
This is another case written by Pendell Meyers (who is helping to edit the blog and has many great recent posts) Case A 45 year old man was driving to work when he experienced acute onset sharp left sided chestpain with paresthesias of the left arm. A repeat ECG was recorded with pain 2/10: Not much change.
They had difficulty describing their symptoms, but complained of severe weakness, nausea, vomiting, headache, and chestpain. They described the chestpain as severe, crushing, and non-radiating. Altogether, this strongly suggests inferolateral OMI, particularly in a patient with acute chestpain.
The patient presented with chestpain. I was taught that the tell-tale sign of ischemia vs an electrical abnormality was in the hx, i.e. chestpain for the ischemia and potential syncope for brugada. Only 5-18% of ED patients with chestpain have a myocardial infarction of any kind. Is it Brugada pattern?
Submitted and written by Megan Lieb, DO with edits by Bracey, Smith, Meyers, and Grauer A 50-ish year old man with ICD presented to the emergency department with substernal chestpain for 3 hours prior to arrival. At this time he reported ongoing chestpain and was given aspirin and nitroglycerin.
Written by Jesse McLaren Two 70 year olds had acute chestpain with nausea and shortness of breath, and called paramedics. Today's patient is a 70-year old adult who called EMS because of new-onset chestpain , associated with nausea and shortness of breath. Who needs the cath lab?
Chris Mondie of the Newark Beth Israel Emergency Medicine Residency sent this case A 50-something man presented with acute chestpain. Defibrillated out of v fib in the cath lab. Here is his ECG: As you can see, the computer called it completely normal What do you think? 100% proximal LAD successfully stented.
He was defibrillated. The formula results in 23.43, just above the 23.4 The patient was diagnosed with esophageal reflux and was being discharged by the nurse when he had a cardiac arrest. Here is his post resuscitation ECG: Now the diagnosis is obvious. Anterolateral STEMI.
While in the hospital, he had witnessed ventricular fibrillation (VF) arrest for which he received external defibrillation. He had minimal in-stent restenosis on angiography but had only minimal cardiac enzyme elevation and did not have antecedent chestpain before either of his events.
He was defibrillated into VT. He then underwent dual sequential defibrillation into asystole. Just as important is pretest probability: did the patient report chestpain prior to collapse? See these related cases: Cardiac arrest, defibrillated, diffuse ST depression and ST Elevation in aVR. They started CPR.
The patient’s chestpain spontaneously resolved before he was evaluated and has a repeat ECG obtained at 22:12 obtained shown below. In context, of course, it is clear that the patient is reperfusing, as pain has dissipated and the diagnostic findings of OMI have become more nonspecific. This ECG is more difficult.
He underwent further standard resuscitation EXCEPT that we applied the Inspiratory Threshold Device ( ResQPod ) AND applied Dual Sequential Defibrillation (this simply means we applied 2 sets of pads, had 2 defib machines, and defibrillated with both with only a fraction of one second separating each defibrillation.
It cannot be treated with a defibrillator and often leads to death. Ventricular fibrillation is a type of irregular heartbeat that can cause the heart to stop beating, but an electric shock from a defibrillator can trigger the beating again.
Patients may feel a fluttering in the chest, chestpain, shortness of breath and dizziness or lightheadedness as a result. The rapid heartbeat may only last for a few seconds, but during that time, the heart is beating so fast that it can’t get enough blood to the rest of the body.
This was written by Magnus Nossen, from Norway, with comments and additions by Smith A 50 something smoker with no previous medical hx contacted EMS due to acute onset chestpain. Upon EMS arrival the patient appeared acutely ill and complained of chestpain. An ECG was recorded immediately and is shown below.
Implantable Cardioverter-Defibrillator (ICD) to help manage dangerous heart rhythms. Coronary Artery Bypass Surgery for those with blocked arteries, improving blood flow to the heart muscle. Left Ventricular Assist Device (LVAD) , a mechanical pump that helps the heart pump blood more effectively.
A 56 yo f with h/o HTN and hypercholesterolemia called EMS from home after onset of L chestpain radiating to the left arm. She was defibrillated successfully from ventricular fibrillation and developed a perfusing rhythm. Before EMS arrived, she had "seizure activity" and became unresponsive. She was intubated.
He was at the gym when he had the onset of chestpain. This patient is 38 years old with hyperlipidemia. EMS was activated and recorded the following ECG (scanned from a prehospital ECG, so the quality is not perfect): Notice the small Q wave in V1 followed by a very large R-wave, with a prolonged QRS. There is a wide S-wave in V6.
He woke up alert and with chestpain which he also had experienced intermittently over the previous few days. The history in today's case with sudden loss of consciousness followed by chestpain is very suggestive of ACS and type I ischemia as the cause of the ECG changes. What do you think?
The patient in today’s case is a previously healthy 40-something male who contacted EMS due to acute onset crushing chestpain. The pain was 10/10 in intensity radiating bilaterally to the shoulders and also to the left arm and neck. He required multiple defibrillations within a period of a few hours. What do you think?
Retrospective questioning of the driver who brought this patient to the hospital revealed that he was having chestpain as the reason for presenting to the ED. A series of VFib episodes followed — each time with successful defibrillation. He became unconscious on arrival. ROSC ( R eturn O f S pontaneous C irculation ).
So this is a typical Brugada syndrome ECG, which can be easily mistaken for an acute myocardial infarction with ST elevation in anterior leads may be taken as STEMI if the person presents with chestpain for some other reason. The importance is that, it is an important cause of inappropriate discharge of defibrillators.
Written by Willy Frick A 57 year old man with was admitted to the hospital with chestpain. But artifact is "alive and well" — and learning to recognize it will amaze many of your colleagues ( and may serve to avoid an unnecessary defibrillation or two ). The rhythm terminated before it could be captured on 12-lead.
The chestpain quickly subsided. The patient was given chest compressions while waiting for the cardiac arrest team to arrive. After about 90 seconds of chest compressions she awoke. She spontaneously converted (Defibrillation was not performed). Are you worried about OMI in this case?
BACKGROUND:There is no specific treatment for sudden cardiac arrest (SCA) manifesting as pulseless electric activity (PEA) and survival rates are low; unlike ventricular fibrillation (VF), which is treatable by defibrillation.
Written by Pendell Meyers and Peter Brooks MD A man in his 30s with no known past medical history was reported to suddenly experience chestpain and shortness of breath at home in front of his family. It was reportedly a PEA arrest; there was no recorded V Fib and no defibrillation. CPR was initiated immediately. This is a quiz.
Written by Pendell Meyers, edits by Steve Smith A man in his 60s with history of hypertension and MI 10 years ago, with PCI, presented to an outside hospital complaining of chestpain that started while mowing the lawn. He was defibrillated immediately and had return of normal mental status.
This patient, who is a mid 60s female with a history of hypertension, hyperlipidemia and GERD, called 911 because of chestpain. A mid 60s woman with history of hypertension, hyperlipidemia, and GERD called 911 for chestpain. She was defibrillated and resuscitated.
People who do have symptoms may experience episodes of fainting, chestpain, shortness of breath or irregular heartbeats. HCM affects approximately 1 in every 500 individuals; however, a significant portion of cases remain undiagnosed because many people do not exhibit symptoms.
There was no chestpain. She was given CRT-D (Cardiac Resynchronization Therapy-Defibrillator). This was written by Magnus Nossen The patient is a female in her 50s. She presented with a one week hx of «dizziness» and weakness. She was feeling fine prior to the last seven days. She did admit to shortness of breath on exertion.
The patient was put on Extracorporeal Life Support in the ED 3 hours after initial resuscitation, the core temp was 30° C and the patient was defibrillated with a single attempt. Although in the context of chestpain such ST depression would be all but diagnostic of posterior OMI, one should make no conclusions in such an unusual case.
She was never defibrillated. A recent similar case: A 40-something with chestpain. As was seen in this case — defibrillation and/or overdrive pacing may be needed. She was given 3 mg IV epinephrine and multiple rounds of ACLS over approximately 20 minutes. What do you think? Is this inferior MI?
He was found in ventricular fibrillation and defibrillated, then brought to a local ED which does not have a cath lab. The patient was defibrillated, and then taken to the nearest ED where ECG #1 was obtained ( Figure-1 ). Here is the initial ED ECG: This is pretty obviously and inferior posterior OMI, right?
It was from a patient with chestpain: Note the obvious Brugada pattern. The elevated troponin was attributed to either type 2 MI or to non-MI acute myocardial injury. There is no further workup at this time. Smith: Here is a case that was just texted to me today from a former resident. This patient ruled out for MI.
She did notice something slightly wrong subjectively, but had no palpitations, chestpain, or SOB, or any other symptom. This middle-aged patient has a remote history of cardiac surgery as a young child for a "heart murmur". Her Apple Watch suddenly told her that she is in atrial fibrillation. She was on no medications. Methods.
12 minutes later, the patient went back into VFib arrest and underwent another 15 minutes of resuscitation followed by successful defibrillation and sustained ROSC. In total, he received approximately 40 minutes of CPR and 7 defibrillation attempts. EMS found the patient in VFib and performed ACLS for 26 minutes then obtained ROSC.
A late middle-aged man presented with one hour of chestpain. At cath, he immediately had incessant Torsades de Pointes requiring defibrillation 7 times and requiring placement of a transvenous pacer for overdrive pacing at a rate of 80. Most recent echo showed EF of 60%. He had recently had a NonSTEMI.
This patient had many complaints including chestpain. Comment by K EN G RAUER, MD ( 2/11 /2023 ): = Today’s case is from a patient with “many complaints”, including chestpain — and, an ECG that raised concern about acute anterior OMI. Treatment is by ICD ( implantable cardioverter defibrillator ).
When a person experiences a heart attack or myocardial infarction, they may feel chestpain and other symptoms in different parts of their body. These issues can only be addressed in an ICCU (Intensive Coronary Care Unit) setting, where temporary pacemakers and defibrillators are available.
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