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This was sent by anonymous The patient is a 55-year-old male who presented to the emergency department after approximately 3 to 4 days of intermittent central boring chestpain initially responsive to nitroglycerin, but is now more constant and not responsive to nitroglycerin. It is unknown when this pain recurred and became constant.
I was working at triage when the medics brought this patient who is 65 yo and has had chestpain for 12 hours. I took the patient to the criticalcare area and questioned him more on the way. The pain had been intermittent until an hour before arrival, when he called 911. It was opened and stented.
Given the presentation, the cardiologist stented the vessel and the patient returned to the ICU for ongoing criticalcare. He did not remember whether he had experienced any chestpain. At his family's request, he was transferred to a hospital closer to his home to continue care.
The 50-something patient with history of coronary stenting and slightly reduced LV ejection fraction. This EKG was recorded as part of a standing order for criticalcare. In the setting of prior stenting and reduced left ventricular ejection fraction, would pursue a heart team revascularization approach Syntax score 28.5,
A 50-something with no previous cardiac history and no risk factors presented to the ED with acute chestpain (pressure) that radiated to the left arm. It was stented with good results. An ECG was immediately recorded: Computer read: Normal ECG What do you think? There is ST depression in V1-V3.
I activated the cath lab and brought her to the criticalcare area. Angiogram showed a distal RCA occlusion which was stented. Echo showed inferior wall motion abnormality.
This middle aged male with h/o GERD but also h/o stents presented to the ED with chestpain. The initial troponin I returned at 1500 ng/L and another ECG was recorded as the patient complained of 9/10 chestpain at 10 hours after the first Now the T-wave in III is fully upright, suggesting re-occlusion.
Submitted and written by Alex Bracey, with edits by Pendell Meyers and Steve Smith: I was walking through the criticalcare section of the ED when I overheard a discussion about the following ECG. The patient was given fentanyl initially for chestpain with minimal effect and then vomited which was followed by zofran and famotidine.
The patient was otherwise healthy, had no past history, and had never had chest discomfort before. I immediately activated the criticalcare team and walked the patient to the criticalcare area, our "Stabilization Room." Opened and stented. There are relatively large T-waves in V4-V6.
A late middle-aged man presented with one hour of chestpain. This was stented. Crit Care Med. 1991 May;19(5):694-9 Objective: To evaluate the efficacy and safety of potassium replacement infusions in critically ill patients. Setting: Multidisciplinary criticalcare unit. The patient stabilized.
Dizziness is so unlikely to be OMI without an obvious ECG, that I am going to pretend that this patient presented with chestpain. So let's pretend this is acute chestpain. 20 cases with pseudonormalization Case continued The patient was moved to the criticalcare area, and cardiology was consulted.
After stent deployment, we often see improvement in the ST-T within seconds or minutes. Here is the final angiogram following placement of a stent in the ostial RCA. 2:04 PM, post stent deployment You can see that even after complete restoration of flow, the ECG still looks terrible, V most of all. link] Jentzer, J. Kashou, A.
1) Very high initial troponin of 45,000 ng/L 2) A full day of chestpain 3) Q-waves on the ECG, with some T-wave inversion Here is one frame of the CT scan which includes the heart: Can you spot the infarct? It was opened and stented. There is STE in V5-6. There are new Q-waves in aVL, V5-6. How do I know?
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