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This EKG was recorded as part of a standing order for criticalcare. 2 days later This is a typical LVH pattern, without ischemia Patient underwent 4 vessel CABG. Upon questioning patient, he denies having any chestpain or chest tightness of any sort. Chestpain is squeezing or tight in nature.
This 54 year old patient with a history of kidney transplant with poor transplant function had been vomiting all day when at 10 PM he developed severe substernal crushing chestpain. He presented to the Emergency Department with a blood pressure of 111/66 and a pulse of 117. He had this ECG recorded.
He was a 30-something with chestpain. A male in his 30's complained of sudden severe substernal chestpain. Perhaps they indicate an open artery with minimal flow and severe subendocardial ischemia, but not total subepicardial ischemia. Here is one case of a patient I saw. But we find that this is unusual.
This middle aged male with h/o GERD but also h/o stents presented to the ED with chestpain. The initial troponin I returned at 1500 ng/L and another ECG was recorded as the patient complained of 9/10 chestpain at 10 hours after the first Now the T-wave in III is fully upright, suggesting re-occlusion.
Submitted and written by Alex Bracey, with edits by Pendell Meyers and Steve Smith: I was walking through the criticalcare section of the ED when I overheard a discussion about the following ECG. The patient was given fentanyl initially for chestpain with minimal effect and then vomited which was followed by zofran and famotidine.
We knew only that the ECG belonged to a man in his 50s with chestpain and normal vitals. We brought the patient into one of our criticalcare rooms and immediately got more history while recording this repeat ECG: The STE in I has greatly diminished or entirely disappeared. No prior available. We activated the cath lab.
This was interpreted by the treating clinicians as not showing any evidence of ischemia. Given the presentation, the cardiologist stented the vessel and the patient returned to the ICU for ongoing criticalcare. He did not remember whether he had experienced any chestpain. He was admitted to cardiology.
A late middle-aged man presented with one hour of chestpain. If there is polymorphic VT with a long QT on the baseline ECG, then generally we call that Torsades, but Non-Torsades Polymorphic VT can result from ischemia alone. Crit Care Med. Setting: Multidisciplinary criticalcare unit.
He denied any chestpain or shortness of breath and stated he felt at his baseline yesterday prior to drug use. They recommended repeating his ECG and awaiting troponin since the patient did not have any chestpain. He complained of generalized weakness and left lower extremity numbness. What is it?
A middle aged patient who was 3 weeks s/p STEMI came from cardiac rehab where he developed some chestpain, dyspnea and weakness on the treadmill. In the ED he had some continued chestpain and hypotension. Here was his ECG: There are inferior and lateral Q-waves with T-wave inversion in the corresponding leads.
Case 1: 20-something woman with chestpain Case 2: 50-something man with chestpain Case 1 A 20-something yo woman presented in the middle of the night with severe crushing chestpain. The blood pressure was 170/100 in the criticalcare area. No ECG was recorded after pain resolution.
1) Very high initial troponin of 45,000 ng/L 2) A full day of chestpain 3) Q-waves on the ECG, with some T-wave inversion Here is one frame of the CT scan which includes the heart: Can you spot the infarct? There is STE in V5-6. There are new Q-waves in aVL, V5-6. SUBACUTE) OMI, that would result in an undesirable delay.
It is possible there is microvascular dysfunction producing residual transmural ischemia. But this is most common when there is prolonged ischemia, and this patient had the fastest reperfusion imaginable! Just a few weeks ago, I took care of a patient who had ostial RCA OMI (TIMI 0 at cath) and his only complaint was syncope!
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