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A middle aged male presented at midnight after 14 hours of constant, severe substernal chestpain, radiating to his throat and to bilateral jaws, and associated with diaphoresis. The pain was not positional, pleuritic, or reproducible. It was not relieved by anything. He had no previous medical history. Is it STEMI or NonSTEMI?
Sinus tachycardia has many potential causes. This is especially true for the elderly patient with sinus tachycardia. What is the cause of the sudden tachycardia? Angiography usually reveals an absence of collateral circulation to the infarct zone. The patient in today’s case suddenly became tachycardic while sleeping.
They had already cardioverted at 120 J, then 200 J, which resulted in the following: Ventricular Tachycardia They then cardioverted at 200 J which r esulted in the same narrow complex rhythm shown above, at 185 beats per minute. This would treat both SVT or sinus tachycardia. I suggested esmolol if the heart rate did not improve.
Chest trauma was suspected on initial exam. The ECG shows sinus tachycardia with RBBB and LAFB, without clear additional superimposed signs of ischemia. Gunshot wound to the chest with ST Elevation Would your radiologist make this diagnosis, or should you record an ECG in trauma? Circulation: Cardiovascular Imaging.
Although the patient reported experiencing mild pressure-like chestpain, there was suspicion among clinicians that this might be indicative of an older change. While the initial impression might not immediately suggest ventricular tachycardia (VT), a closer examination raises suspicion. What is the rhythm?
Circulation, Volume 150, Issue Suppl_1 , Page A4119267-A4119267, November 12, 2024. Advanced cardiac imaging especially in atypical presentations, can aid in early diagnosis.Case:A 59 year-old man with history of biopsy-proven pulmonary sarcoidosis presented with non exertional chestpain for 2 months.
Circulation: Arrhythmia and Electrophysiology, Ahead of Print. Postablation chestpain consistent with pericarditis was reduced with colchicine (4% versus 15%; HR, 0.26 [95% CI, 0.09–0.77];P=0.02) BACKGROUND:Inflammation may promote atrial fibrillation (AF) recurrence after catheter ablation. 2.02];P=0.89). 1.99];P=0.55).CONCLUSIONS:Colchicine
A middle aged male with no h/o CAD presented with one week of crescendo exertional angina, and had chestpain at the time of the first ECG: Here is the patient's previous ECG: Here is the patient's presenting ED ECG: There is isolated ST depression in precordial leads, deeper in V2 - V4 than in V5 or V6. There is no ST elevation.
This strip was obtained: Apparent Wide Complex Tachycardia at a rate of 280 What do you think? Troponins 34>33>43, likely secondary to myocardial injury from tachycardia. Sinus tachycardia does not go this fast. A 60-something ow healthy male had syncope while on treadmill. What do you want to do?
POTS stands for postural orthostatic tachycardia syndrome. Let’s call it Postural Orthostatic Tachycardia Syndrome – that’s not really a diagnosis – it’s just a medical jargon filled term for what the patient has just told us. If we can therefore increase the circulating volume, then patients do feel better.
Written by Colin Jenkins and Nhu-Nguyen Le with edits by Willy Frick and by Smith A 46-year-old male presented to the emergency department with 2 days of heavy substernal chestpain and nausea. The patient continued having chestpain. Circulation Research , 56 (2), 184–194. Is there STEMI? What is the rhythm?
It was edited by Smith CASE : A 52-year-old male with a past medical history of hypertension and COPD summoned EMS with complaints of chestpain, weakness and nausea. Circulation. Circulation 67, No. Circulation 1970;41:623-627 9. The paramedic’s initial impression of the patient was that he was critically ill.
Case submitted and written by Dr. Mazen El-Baba and Dr. Emily Austin, with edits from Jesse McLaren A 50 year-old patient presented to the Emergency Department with sudden onset chestpain that began 14-hours ago. The pain improved (6/10) but is persisting, which prompted him to visit the Emergency Department. What do you think?
She denied chestpain and denied feeling any palpitations, even during her triage ECG: What do you think? She was awake, alert, well perfused, with normal mental status and overall unremarkable physical exam except for a regular tachycardia, possible rales at both bases, some mild RUQ abdominal tenderness.
There was no chestpain. Shortly after isoprenalin infusion was initiated, there were short runs of ventricular tachycardia. The physiologic reason for this — is thought to be the result of momentarily increased circulation from mechanical contraction arising from the "sandwiched in" QRS complex.
This middle-aged man with no cardiac history but with significant history of methamphetamin and alcohol use presented with chestpain and SOB, worsening over days, with orthopnea. Here was his ED ECG: There is sinus tachycardia (rate about 114) with nonspecific ST-T abnormalities. BP:143/99, Pulse 109, Temp 37.2 °C
The patient in today’s case is a previously healthy 40-something male who contacted EMS due to acute onset crushing chestpain. The pain was 10/10 in intensity radiating bilaterally to the shoulders and also to the left arm and neck. Written By Magnus Nossen — with edits by Ken Grauer and Smith. The below ECG was recorded.
A late middle-aged man presented with one hour of chestpain. See here for management of Polymorphic Ventricular Tachycardia , which includes Torsades. mEq of K pushed fast and circulated theoretically would raise serum K immediately by 1.0 Most recent echo showed EF of 60%. He had recently had a NonSTEMI.
Below are 6 anecdotal cases of true complete left main occlusion with no collateral circulation: 3 have STE in aVR 1 has no ST shift in aVR 2 have STD in aVR The ECG can have a variety of presentations in LM Occlusion. You'll see that there is collateral circulation from the RCA. This is her ECG: An obvious STEMI, but which artery?
Otherwise vitals after intubation were only notable for tachycardia. An initial EKG was obtained: Computer read: sinus tachycardia, early acute anterior infarct. It was from a patient with chestpain: Note the obvious Brugada pattern. Circulation, 117, 1890–1893. [3]: There is no further workup at this time.
Answer : you must treat the patient's underlying condition causing sinus tachycardia, and repeat the ECG at the lower heart rate. Optimal QT interval correction formula in sinus tachycardia for identifying cardiovacular and mortality risk: Findings from the Penn Atrial Fibrillation Free study. which is 0.6 So is it really prolonged?
When a person experiences a heart attack or myocardial infarction, they may feel chestpain and other symptoms in different parts of their body. This indicates that restoring normal blood circulation as quickly as possible will result in less damage. So, how do you recognize a heart attack? Perform rescue breaths twice.
Check : [vitals, SOB, ChestPain, Ultrasound] If the patient has Abdominal Pain, ChestPain, Dyspnea or Hypoxemia, Headache, Hypotension , then these should be considered the primary chief complaint (not syncope). Circulation. Most physicians will automatically be worried about these symptoms.
A 69 year old woman with a history of hypertension presented to the emergency department by EMS for evaluation of chestpain and shortness of breath. She awoke in the morning with sharp chestpain which worsened throughout the morning. As her pain worsened, so did her dyspnea. This was written by Hans Helseth.
A middle-age woman with no previous cardiac history called 911 for chestpain. Return of spontaneous circulation (ROSC) was the primary outcome. This was her prehospital ECG: What do you think? There is sinus rhythm with RBBB and obvious LAD OMI (proximal LAD occlusion): hyperacute T-waves in I, aVL and minimal STE in V1, V2.
He had no chestpain, dyspnea, or any other anginal equivalent, and his vital signs were normal. Circulation Research , 114 (12), 18521866. Circulation , 92 (3), 657671. Circulation , 125 (3), 491496. Circulation , 145 (13), 10021019. Journal of Geriatric Cardiology , 19 (6). link] Bentzon, J. Virmani, R.,
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