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What does the angiogram show? The Echo? The CT coronary angiogram? How do you explain this?

Dr. Smith's ECG Blog

Angiogram No obstructive epicardial coronary artery disease Cannot exclude non-ACS causes of troponin elevation including coronary vasospasm, stress cardiomyopathy, microvascular disease, etc. We know that most type 1 acute MI due to plaque rupture and thrombosis occurs in lesions that are less than 50% (see Libby reference).

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ECG Blog #415 — The Cath showed NO Occlusion!

Ken Grauer, MD

Non-obstructive coronary disease at the time cardiac cath is done does not necessarily imply there was no plaque rupture with thrombus. These plaques will often not be recognized as "culprits" — because no fissuring or ulceration is seen. Longterm prognosis of patients with MINOCA clearly depends on the underlying etiology.

Blog 163
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Concerning EKG with a Non-obstructive angiogram. What happened?

Dr. Smith's ECG Blog

The commonest causes of MINOCA include: atherosclerotic causes such as plaque rupture or erosion with spontaneous thrombolysis, and non-atherosclerotic causes such as coronary vasospasm (sometimes called variant angina or Prinzmetal's angina), coronary embolism or thrombosis, possibly microvascular dysfunction. It is not rare.

Plaque 127
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"A patient just arrived as a transfer for NSTEMI."

Dr. Smith's ECG Blog

Given her lack of risk factors for coronary disease, and the fact that she was a 53 year old woman with compatible history and echo findings, stress cardiomyopathy rose to the top of my differential. Of course, stress cardiomyopathy is a diagnosis of exclusion. But not all OMI is atherosclerotic in nature.

SCAD 124
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SBC Scientific Publication Award – 2024

International Journal of Cardiovascular Sciences

Awards granted: a) Plaque for each awarded article. The corresponding recognition will entitle the honoree to a plaque. The Award will correspond to articles published in 2023. b) Registration for the next subsequent congress. c) Credit of R$ 2,000.00 for enrollment in courses at the Heart University. Except for postgraduate courses.

Article 52
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Wide Complex Tachycardia

EMS 12-Lead

I interpreted the ECG as VT with two primary etiological possibilities: 1. Abrupt plaque ulceration of Type 1 ACS leading to VT. 2. Baseline fibrotic substrate from dilated cardiomyopathy leading to VT. From a clinical standpoint I reconciled the first possibility as most probable since the chest discomfort came first.

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Upon arrival to the emergency department, a senior emergency physician looked at the ECG and said "Nothing too exciting."

Dr. Smith's ECG Blog

by making it clear to everyone that this is NOT an EKG that one sees with takotsubo cardiomyopathy. Hospital Course The patient was taken emergently to the cath lab which did not reveal any significant coronary artery disease, but she was noted to have reduced EF consistent with Takotsubo cardiomyopathy. It can only be seen by IVUS.

Plaque 52