This site uses cookies to improve your experience. To help us insure we adhere to various privacy regulations, please select your country/region of residence. If you do not select a country, we will assume you are from the United States. Select your Cookie Settings or view our Privacy Policy and Terms of Use.
Cookie Settings
Cookies and similar technologies are used on this website for proper function of the website, for tracking performance analytics and for marketing purposes. We and some of our third-party providers may use cookie data for various purposes. Please review the cookie settings below and choose your preference.
Used for the proper function of the website
Used for monitoring website traffic and interactions
Cookie Settings
Cookies and similar technologies are used on this website for proper function of the website, for tracking performance analytics and for marketing purposes. We and some of our third-party providers may use cookie data for various purposes. Please review the cookie settings below and choose your preference.
Strictly Necessary: Used for the proper function of the website
Performance/Analytics: Used for monitoring website traffic and interactions
Background High-intensity physical activity has traditionally been discouraged in patients with hypertrophic cardiomyopathy due to concerns about triggering suddencardiacdeath. However, current guidelines adopt a more liberal stance, and evidence on risk factors for exercise-related suddencardiacdeath remains limited.
These numbers indicate rising participation — an increase in people involved in chronic endurance exercise training for the sake of competition and health. A list of the causes of death reveals an underlying pattern. Many, but not all, are cardiac-related issues (i.e., ” Isn’t exercise good for us? .”
Although the 2024 ACC/American Heart Association (AHA) and 2023 European Society of Cardiology (ESC) guidelines stratify well overall the risk of suddencardiacdeath (SCD) in patients with hypertrophic cardiomyopathy (HCM).
Hypertrophic cardiomyopathy (HCM)-related suddencardiacdeath (SCD) rates with contemporary management are low; however, high-intensity exercise can induce fatal arrhythmias in HCM patients. Thus, current guidelines recommend avoiding high-intensity exercise in HCM patients at high risk for SCD1,2.
Maron, MD “Our findings provide enthusiasm that a novel drug therapy with ninerafaxstat may provide nonobstructive HCM patients an opportunity to achieve a better quality of life by decreasing symptom burden and improving exercise capacity,” said Martin S. For more information on hypertrophic cardiomyopathy, visit CardioSmart.org/HCM.
The American College of Cardiology (ACC) and the American Heart Association (AHA) today released a new clinical guideline for effectively managing individuals diagnosed with hypertrophic cardiomyopathy (HCM). Ommen, MD, FACC , medical director of the Mayo Hypertrophic Cardiomyopathy Clinic and chair of the guideline writing committee.
Cardiovascular mortality, driven by suddencardiacdeath, is the main reason for dying while waiting for heart transplantation (HTx). Aims Timely referrals for transplantation and left ventricular assist device (LVAD) play a key role in favourable outcomes in patients with advanced heart failure (HF).
Director of the Hypertrophic Cardiomyopathy Center at the Lahey Hospital and Medical Center. These data elaborate on the primary results from SEQUOIA-HCM and further inform the relevance to clinical practice of aficamten as a next-in-class cardiac myosin inhibitor for adult patients with obstructive HCM.” References: Maron M, et al.
Sent by anonymous, written by Pendell Meyers A male in his teens presented with complaints of chest discomfort and dyspnea beginning while exercising but without obvious injury. He immediately stopped exercising and symptoms started to improve. Pattern consistent with Takotsubo's cardiomyopathy." No similar symptoms in the past.
Suddencardiacdeath in cardiomyoptahies: incidence, risk factors and prevention. Significant LGE at CMR; LVEF <50%; abnormal blood pressure response during exercise test; LV apical aneurysm; high-risk genotype. However, unresolved issues still challenge the guidance of SCD prevention in patients with cardiomyopathies.
BACKGROUND:Exercise-induced cardiac remodeling can be profound, resulting in clinical overlap with dilated cardiomyopathy, yet the significance of reduced ejection fraction (EF) in athletes is unclear. Circulation, Ahead of Print. During follow-up, no athletes developed symptomatic heart failure or arrhythmias.
We organize all of the trending information in your field so you don't have to. Join thousands of users and stay up to date on the latest articles your peers are reading.
You know about us, now we want to get to know you!
Let's personalize your content
Let's get even more personalized
We recognize your account from another site in our network, please click 'Send Email' below to continue with verifying your account and setting a password.
Let's personalize your content