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Specific genetic variants, such as those affecting cholesterol metabolism, can increase the likelihood of plaque buildup in the arteries. Cardiomyopathies: These diseases affect the heart muscle, impairing its ability to pump blood effectively. Heart imaging, such as echocardiograms or CT scans.
Coronary Artery Disease (CAD) CAD, which involves the narrowing or blockage of coronary arteries due to plaque buildup, can reduce blood flow to the heart. CardiomyopathyCardiomyopathy is a condition that affects the heart muscle, causing it to become enlarged, thick, or rigid.
Example here: DIffuse ST Elevation with Apical Ballooning: is it Takotsubo Stress Cardiomyopathy? Mild Plaque no angiographically significant obstructive coronary artery disease. Stress induced cardiomyopathy (Takotsubo like LV dysfunction) possible. Therefore, the cath lab was activated. The initial hs troponin was = 41 ng/L.
I interpreted the ECG as VT with two primary etiological possibilities: 1. Abrupt plaque ulceration of Type 1 ACS leading to VT. 2. Baseline fibrotic substrate from dilated cardiomyopathy leading to VT. Corresponding echocardiogram demonstrated LV systolic dysfunction with an EF 30%.
The commonest causes of MINOCA include: atherosclerotic causes such as plaque rupture or erosion with spontaneous thrombolysis, and non-atherosclerotic causes such as coronary vasospasm (sometimes called variant angina or Prinzmetal's angina), coronary embolism or thrombosis, possibly microvascular dysfunction. It is not rare.
by making it clear to everyone that this is NOT an EKG that one sees with takotsubo cardiomyopathy. Hospital Course The patient was taken emergently to the cath lab which did not reveal any significant coronary artery disease, but she was noted to have reduced EF consistent with Takotsubo cardiomyopathy. It can only be seen by IVUS.
If our valves are leaky then again, a smaller volume of blood is effectively ejected because some leaks back Cardiomyopathies – if the heart muscle itself is defective in some way then that makes it weaker. This includes conditions like familial hypertrophic cardiomyopathy, familial dilated cardiomyopathy etc.
However, an echocardiogram is a different test, also conducted for heart activity. Electrocardiogram, echocardiogram, and some other tests are done for patients with cardiac arrest. An ECG machine is able to detect other abnormalities of the heart as well, such as hypertrophic cardiomyopathy or overly thick heart muscles.
The combination of prolonged QT and deep T wave inversion throughout the precordium is typical of Takotsubo syndrome, or Stress Cardiomyopathy – which can occur in the context of a physiologically distressed ICU patient, further compromising their hemodynamics. The coronary angiogram revealed no critical stenosis, or acute plaque ulceration.
So cardiomyopathies, valve problems, myocarditis and previous heart attacks all cause a problem with the pumping function of the heart. Overall though a normal cardiac MRI is even more reassuring than a normal echocardiogram. This is an ultrasound (a bit like the type that we use on pregnant women to look at the baby).
These are reperfusion T-waves (the same thing as Wellens' waves) Echocardiogram Regional wall motion abnormality-distal septum and apex. Troponins may be negative with very rapid reperfusion, or measured too late, or chronically elevated due to cardiomyopathy or renal failure. Regional wall motion abnormality-distal inferior wall.
Whenever I see PVCs with the morphology and axis seen in todays case I always look for signs of AC ( Arrhythmogenic Cardiomyopathy ). Arrhythmogenic cardiomyopathy often manifests with PVCs from the RV. The ECG in Figure-1 however, shows no signs of arrhythmogenic cardiomyopathy. There were no plaques or stenoses.
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