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Background Hypertrophic cardiomyopathy (HCM) is commonly associated with atrial fibrillation (AF), but its impact on outcomes in real-world practice is uncertain. years) were enrolled in the EURObservational Research Programme (EORP) Cardiomyopathy/Myocarditis Registry. Methods Overall, 1739 adult patients with HCM (40.9%
Better risk stratification is needed to evaluate patients with non-ischemic cardiomyopathy (NICM) for prophylactic implantable cardioverter-defibrillators (ICD). Growing evidence suggests cardiac magnetic resonance imaging (CMR) may be useful in this regard.
He was defibrillated, but they also noticed that he was being internally defibrillated and then found that he had an implantable ICD. He was unidentified and there were no records available After 7 shocks, he was successfully defibrillated and brought to the ED. Bedside US shows extremely poor EF with dilated cardiomyopathy.
In contrast to primary prevention implantable cardioverter defibrillators (ICD) in ischemic cardiomyopathy, the data supporting ICD placement in patients with non-ischemic cardiomyopathy (NICM) is less robust. Moreover, there is scarce data about risk factors for appropriate and inappropriate therapy in diverse populations.
Cardiac sarcoidosis (CS), a rare condition characterized by non-caseating granulomas, can manifest with symptoms such as atrioventricular block and ventricular tachycardia (VT), as well as mimic inherited cardiomyopathies. A 58-year-old woman presented with sustained VT with a prior diagnosis of hypertrophic cardiomyopathy (HCM).
Implantable cardioverter defibrillators (ICD) have proved their favourable outcomes on survival in selected patients with cardiomyopathy. However, recent studies have questioned the protective role of ICD in non-ischemic cardiomyopathy (NICM) for primary prevention.
Chagasic cardiomyopathy has been associated with high defibrillator shock rates and poor clinical outcomes. The benefits of VT ablation in patients with Chagasic cardiomyopathy have been evaluated in only a few studies. Chagas disease is an endemic condition in Latin America.
And of course Ken's comments at the bottom) An elderly obese woman with cardiomyopathy, Left bundle branch block, and chronic hypercapnea presented hypoxic with altered mental status. I do not see OMI here and all trops were only minimally elevated, consistent with either chronic injury from cardiomyopathy or with acute injury from sepsis.
Wide complex tachycardia was seen on ILR tracings, and he was presumptively diagnosed with non-ischemic cardiomyopathy (NICM) and ventricular tachycardia (VT). Implantable cardioverter-defibrillator (ICD) was implanted by his local hospital given syncope.
Clinical introduction A patient in his 60s with a history of ischaemic cardiomyopathy and severely reduced left ventricular ejection fraction of 20% presents with worsening dyspnoea on exertion. mL/kg/min) despite maximal effort.
AimsThis retrospective cohort study aimed to investigate the efficacy of dual-chamber left Bundle branch pacing (LBBP) as an alternative therapy for heart failure patients with complete left bundle branch block (CLBBB) and indications for defibrillator with cardiac resynchronization therapy (CRT-D).Methods34 to 61.06.0% (P<0.001).
An 18-year-old male with arrhythmogenic cardiomyopathy was referred for catheter ablation of ventricular tachycardia after experiencing appropriate implantable cardioverter-defibrillator (ICD) shock. Six months prior, he had undergone uncomplicated implantation of an extravascular ICD (EV-ICD) [1].
T-wave oversensing (TWO) in patients (pts) with a subcutaneous implantable cardioverter defibrillator (S-ICD) has been well described, but P-wave oversensing (PWO) is less common.
Hypertrophic cardiomyopathy (HCM) predisposes patients to cardiac arrest (CA). While established major risk factors may warrant the need for primary prevention by implantable cardioverter-defibrillator (ICD), it is unknown if specific electrocardiographic predictors increase the risk of CA.
He was defibrillated into VT. He then underwent dual sequential defibrillation into asystole. VF should make you think of ischemia, cardiomyopathy (especially scar from old MI), or one many other cardiac but non-ischemic etiologies. This patient was witnessed by bystanders to collapse. They started CPR. sodium bicarbonate.
The efficacy of implantable cardioverter-defibrillators (ICD) in patients with a non-ischaemic cardiomyopathy (NICM) is being debated. Deep Learning models enable feature extraction from high-dimensional data, such as cardiac MRI (CMR) and ECG.
Ventricular tachycardia (VT) increases morbidity and mortality in patients with cardiomyopathy. Catheter ablation (CA) lowers VT recurrence and implantable cardioverter defibrillator (ICD) interventions, however, its effect on all-cause mortality remains unclear.
The extravascular implantable cardioverter-defibrillator (EV-ICD, Medtronic) recently received FDA approval. The pivotal EV-ICD trial recruited primarily patients with cardiomyopathy (84%). Less than 1% of patients enrolled had Brugada syndrome (BrS).
She spontaneously converted (Defibrillation was not performed). Most such rhythms in the setting of ischemia are VF and will not convert without defibrillation. There was hyperkinesis of the basal segments and findings were interpreted as typical of takotsubo cardiomyopathy. At this time the following blood test were available.
Timely diagnosis has significant implications for clinical management, allowing for disease-modifying therapy or implantable cardioverter-defibrillator (ICD) insertion. Conduction system disturbance may represent an early manifestation of underlying structural heart disease, including infiltrative disorders such as sarcoidosis.
Left ventricular assist device (LVAD) patients are at high risk for ventricular tachycardia (VT) due to arrhythmogenic substrate from underlying cardiomyopathy as well as the LVAD inflow cannula. VT is often well tolerated hemodynamically in the presence a LVAD; therefore, antiarrhythmic drugs are first-line for therapy.
Two sisters, diagnosed four days apart with a serious heart condition, explain how hypertrophic cardiomyopathy (and having defibrillators implanted) has changed their lives in surprising ways.
He had a background of arrhythmic right ventricular cardiomyopathy. He had previously undergone the placement of a dual-chamber implantable cardioverter-defibrillator for atrioventricular block and ventricular tachycardia (VT). Clinical introduction A man in his 50s presented to the emergency department with palpitations.
Accurate risk stratification allows for timely implantation of intracardiac defibrillators (ICD) and is currently performed using a multimodality prediction model. (Arg14del) variant carriers are at risk of developing malignant ventricular arrhythmias (MVA).
It is commonly encountered in infiltrative cardiomyopathies but may be associated with myocarditis. Options include a ‘watch-and-wait’ policy, anti-arrhythmic drugs, consideration of a permanent pacemaker or, alternatively, a wearable or implantable cardioverter-defibrillator (ICD).
In patients with heart failure (HF) undergoing left bundle branch area pacing (LBBAP) for cardiac resynchronization therapy (CRT) without a defibrillator (e.g., elderly patients or non-ischemic cardiomyopathy, in whom the use of a defibrillator has been questioned),1 the use of a right ventricular (RV) lead may not be necessary.
After resuscitation and defibrillation , there were no more episodes of TdP. Below is the patient’s 12 lead ECG following defibrillation. The patient was diagnosed with stress cardiomyopathy. Widespread T wave inversions and prolongation of the QT interval is not uncommon in Takotsubo cardiomyopathy.
We describe a case of ventricular pacemaker spikes delivered on the T wave causing PMVT.Case:A 53-year-old female with CAD s/p stent, postpartum cardiomyopathy s/p Bi-V CRT-D (Boston Scientific G124), and paroxysmal atrial fibrillation presented for elective endoscopy and colonoscopy to evaluate her dysphagia and abdominal pain.
She was successfully revived after several rounds of ACLS including defibrillation and amiodarone. The most important clinical entity associated with motion alternans is large pericardial effusion — though motion alternans has also been observed in some cases of hypertrophic cardiomyopathy.
Today's case reminds us of the intuitive logic that if a patient has a shockable arrest ( ie, VFib ) — and following successful defibrillation shows evidence of acute OMI ( even if STEMI criteria are not necessarily fulfilled ) — that such patients have much to gain from immediate cath with PCI. (
vs. 28.8%, p < 0.001), more often had ischeemic cardiomyopathy (49.8% 1.70) and implantable cardioverter-defibrillator (ICD) (multivariable HR 1.40, 95% CI 1.01–1.95). Multivariable Cox regression and propensity score matching (PSM) analyses were performed. ml/min/1.73 m ml/min/1.73 m 76.5], p < 0.001).
We can, therefore, put down the defibrillation pads, set aside the amiodarone, and look further at the ECG. Indeed, bedside Echocardiogram revealed severe left ventricular impairment of Takotsubo cardiomyopathy. Then, the remaining ECG interpretation identified long QT, and T wave inversions, from cardiomyopathy.
The deep learning algorithm helps segment the left ventricle predicting cardiomyopathy and ejection fraction. Along with it, a guideline-based algorithm helps identify implantable cardioverter defibrillators.
She was defibrillated and resuscitated. by making it clear to everyone that this is NOT an EKG that one sees with takotsubo cardiomyopathy. The impact of ST-segment elevation on the prognosis of patients with Takotsubo cardiomyopathy. Smith: this ECG and clinical presentation is diagnostic of LAD Occlusion.
This randomized clinical trial assesses whether cardioverter-defibrillator implantation is more effective than amiodarone therapy for the primary prevention of all-cause mortality and secondary prevention of sudden cardiac death, hospitalization for heart failure, and use of a pacemaker among patients with chronic Chagas cardiomyopathy.
Comprehensive risk assessment combined with shared decision making around the decision to participate in sports seems to be the way of the future, as more fully discussed in the most recent ACC/AHA Guidelines.
The goal of the CHAGASICS trial was to evaluate implantable cardioverter-defibrillators (ICDs) compared with amiodarone among patients with chronic Chagas cardiomyopathy.
The transvenous implanted cardioverter defibrillator (TV-ICD) has been utilized in hypertrophic cardiomyopathy (HCM) for prevention of sudden death for >25 years. More recently, the subcutaneous ICD (S-ICD) has been used for primary prevention in select HCM patients.
CardiomyopathyCardiomyopathy is a condition that affects the heart muscle, causing it to become enlarged, thick, or rigid. Excessive Alcohol or Drug Use Long-term abuse of alcohol or certain drugs can weaken the heart muscle, resulting in cardiomyopathy and eventually cardiomegaly.
Hypertrophic cardiomyopathy (HCM) is associated with risk of sudden cardiac death (SCD). Since approval in late 2012, the subcutaneous implantable cardioverter-defibrillator (SICD) has been used as an alternative to the traditional transvenous ICD (TV-ICD) for SCD prevention in HCM.
Current guidelines for primary prevention implantable cardioverter defibrillator (ICD) placement require persistent left ventricular dysfunction despite optimal therapy and a reasonable expectation of meaningful survival for >1 year for the prevention of sudden cardiac death.
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