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A 70-something female with no previous cardiac history presented with acute chestpain. She awoke from sleep last night around 4:45 AM (3 hours prior to arrival) with pain that originated in her mid back. She stated the pain was achy/crampy. Over the course of the next hour, this pain turned into a pressure in her chest.
The patient presented due to chestpain that was typical in nature, retrosternal and radiating to the left arm and neck. He denied any exertional chestpain. It is unclear if the patient was pain free at this time. He has a medical hx notable for hypertension, hyperlipidemia and previous tobacco use disorder.
Submitted by anonymous, written by Pendell Meyers A woman in her 50s presented to the Emergency Department with chestpain and shortness of breath that woke her from sleep, with diaphoresis. See these other cases of arterial pulse tapping artifact: A 60 year old with chestpain Are these Hyperacute T-waves?
No prior exertional complaints of chestpain, dizziness, lightheadedness, or undue shortness of breath. No family history of sudden cardiac death, cardiomyopathy, premature CAD, or other cardiac issues. He denied headache or neck pain associated with exertion. Pattern consistent with Takotsubo's cardiomyopathy."
Written by Pendell Meyers, edits by Smith and Grauer A man in his late 20s with history of asthma presented to the ED with a transient episode of chestpain and shortness of breath after finishing a 4-mile run. Ct coronaryangiogram showed normal coronary arteries. Hypertrophic Cardiomyopathy or Normal ("Variant")?
Whether you’re gearing up for your Family Medicine or Cardiology Board Exam, you’ll need to master the topic of Hypertrophic Cardiomyopathy (HCM). He has never had any chestpain. Hypertrophic cardiomyopathy is one of them. Hypertrophic cardiomyopathy is one of them. He takes occasional multivitamins.
She reports a known history of Hypertrophic Cardiomyopathy (HCM) with left ventricular outflow tract obstruction and is on daily beta blocker therapy. Cardiology felt her chestpain to be, most likely, the result of coronary supply-demand mismatch in the context of HCM endothelial remodeling (i.e. References Naidu, S.
CT coronaryangiogram — No obstructive coronary disease. Subsequent testing supported the presumption of apical cardiomyopathy as the cause of this patient's sustained VT. CT coronaryangiogram showed no obstructive coronary disease. That said — I was not 100% certain about this interpretation.
He was admitted for monitoring, as his risk of a ventricular dysrhythmia as cause of the syncope is high ( very high due to HFrEF and ischemic cardiomyopathy ). He denied chestpain or dyspnea throughout. Discussion Thus, no further ECGs were recorded and there was no angiogram or stress test or CT coronaryangiogram.
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