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What is the preferred order of vasopressors and ionotropes in the management of cardiogenicshock? How can we best pick up occult cardiogenicshock before it floured shock kicks in? The post Ep 164 CardiogenicShock Simplified appeared first on Emergency Medicine Cases.
Smith comment: This patient did not have a bedside ultrasound. Had one been done, it would have shown a feature that is apparent on this ultrasound (however, this patient's LV function would not be as good as in this clip): This is recorded with the LV on the right. What should be done? Should the cath lab be activated?
24: Joint American College of Cardiology/Journal of the American College of Cardiology Late-Breaking Clinical Trials (Session 402) Saturday, April 6 9:30 – 10:30 a.m.
Just prior to transport, the patient became confused and agitated and, although blood pressure and pulse were OK, I was worried about cardiogenicshock. Angiogram Left main: Severe calcific stenosis of ostial and distal left main. LAD: large caliber vessel with severe calcific stenosis of the proximal LAD with TIMI2 flow.
History sounds concerning for ACS (could be critical stenosis, triple vessel), but differential also includes dissection, GI bleed, etc. 2 cases of Aortic Stenosis: Diffuse Subendocardial Ischemia on the ECG. An elderly man with sudden cardiogenicshock, diffuse ST depressions, and STE in aVR Literature 1. Left main?
Angiography : LMCA — 90-99% osteal stenosis. LCx — 50-69% stenosis of the 1st marginal branch; with 100% distal LCx occlusion. The patient in today’s case presented in cardiogenicshock from proximal LAD occlusion, in conjunction with a subtotally stenosed LMCA. . RCA — 100% proximal occlussion.
Why is the patient in shock? He was in profound cardiogenicshock. They did not have an ultrasound on the ambulance (some local crews are starting to utilize POC limited US in our service areas). There is an obvious inferior STEMI, but what else? This STE is diagnostic of Right Ventricular STEMI (RV MI).
Tachycardia is unusual for OMI, unless the patient is in cardiogenicshock (or getting close). A bedside ultrasound should be done to assess volume and other etiologies of tachycardia, but if no cause of type 2 MI is found, the cath lab should be activated NOW. We can see enough to make out that the rhythm is sinus tachycardia.
Whenever there is tachycardia, I am skeptical of OMI unless it has led to severely compromised ejection fracction with cardiogenicshock. I suspect pulmonary edema, but we are not given information on presence of B-lines on bedside ultrasound, or CXR findings. What "initiates" the aortic stenosis cascade?
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