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This is a value typical for a large subacute MI, n ormal value 48 hours after myocardialinfarction is associated with Post-Infarction Regional Pericarditis ( PIRP ). Sinus tachycardia has many potential causes. This is especially true for the elderly patient with sinus tachycardia.
Because of the tachcardia, I would expect her to be very poor left ventricular function and maybe Cardiogenicshock. Alternatively , it is someone who has an old myocardialinfarction and is now very sick with something else. Still Irregular Blood pressure during these rhythms was adequate; there was no shock.
There is sinus tachycardia. Sinus tachycardia, which exaggerates ST segments and implies that there is another pathology. I have always said that tachycardia should argue against acute MI unless there is cardiogenicshock or 2 simultaneous pathologies. Here is that ECG: What do you think?
Authors' commentary: Cardiogenicshock in the setting of severe aortic stenosis. This patient’s severe aortic stenosis (AS) and associated severe cardiogenicshock likely created the ECG pattern, resulting in a very difficult challenge for our inpatient team. If you can use Doppler, then you can diagnose it.
New insights into the use of the 12-lead electrocardiogram for diagnosing acute myocardialinfarction in the emergency department. An elderly man with sudden cardiogenicshock, diffuse ST depressions, and STE in aVR Literature 1. A slightly prolonged QTc ( although this is difficult to assess given the tachycardia ).
The patient in today’s case presented in cardiogenicshock from proximal LAD occlusion, in conjunction with a subtotally stenosed LMCA. Troponin T peaked at 38,398 ng/L ( = a very large myocardialinfarction, but not massive-- thanks to the pre-PCI spontaneous reperfusion, and rapid internvention!! ).
Assessment was severe sudden cardiogenicshock. Clin Chem [Internet] 2020;Available from: [link] Smith mini-review: Troponin in Emergency Department COVID patients Cardiac Troponin (cTn) is a nonspecific marker of myocardial injury. MyocardialInfarction Type 2 and Myocardial Injury. What is it?
The axiom of "type 1 (ACS, plaque rupture) STEMIs are not tachycardic unless they are in cardiogenicshock" is not applicable outside of sinus rhythm. 2) Tachycardia to this degree can cause ST segment changes in several ways. Sometimes you must correct the rhythm to see what lies underneath. Is this inferor STEMI?
There is sinus tachycardia (do not be fooled into thinking this is VT or another wide complex tachycardia!) This pattern is essentially always accompanied by cardiogenicshock and high rates of VT/VF arrest, etc. The patient arrived to the ED in cardiogenicshock but awake. Code STEMI was activated.
At the bottom of the post, I have re-printed the section on aVR in my article on the ECG in ACS from the Canadian Journal of Cardiology: New Insights Into the Use of the 12-Lead Electrocardiogram for Diagnosing Acute MyocardialInfarction in the Emergency Department Case 1. Widimsky P et al.
Why is the patient in shock? He was in profound cardiogenicshock. Both of these features make inferior + RV MI by far the most likely ( Pseudoanteroseptal MI is another name for this ) There is also sinus bradycardia and t he patient is in shock with hypotension. There is an obvious inferior STEMI, but what else?
When one of these arteries becomes completely blocked by a blood clot, it results in a heart attack, also known as MI (Myocardialinfarction). When a person experiences a heart attack or myocardialinfarction, they may feel chest pain and other symptoms in different parts of their body.
1) as far as I can tell, there is very little data on amiodarone for this indication 2) amiodarone has beta blockade effects which could be deleterious in a patient with large anterior MI with pulmonary edema and at risk for cardiogenicshock (and she did go into shock. DOI: 10.1016/j.resuscitation.2025.110515
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